what's the scientific name for rabies and yellow fever?
537754
Rabies is hydrophobia (from an inability to drink water) and yellow fever is yellow fever, from the jaundice it causes, turning the person yellow
135058
You can find the information to this website:\n\nhttp://en.wikipedia.org/wiki/Rabies
838106
Rabies is spread through saliva or blood from an infected agent. The strain must enter the bloodstream.
999178
The mosquito species that transmits the Chikungunya virus is the Aedes albopictus, commonly known as the Asian tiger mosquito or forest mosquito. There being no vaccine or preventive drug for chikungunya at present, the best way to avoid the infection is to control the mosquito population in the region and prevent mosquito bites.\nhttp://curezone.com/blogs/m.asp?f=380&i=8\n\nLast year, several members of the Iowa State University Department of Entomology presented the results of a study on common catnip. Among their conclusions was the fact that an essential oil in catnip is 10 times more effective at repelling mosquitoes than potent chemicals such as DEET.\nhttp://curezone.com/blogs/m.asp?f=634&i=13\n\nThe symptoms of chikungunya include fever which can reach 39°C, (102.2 °F) a petechial or maculopapular rash usually involving the limbs and trunk, and arthralgia or arthritis affecting multiple joints which can be debilitating. There can also be headache, conjunctival injection and slight photophobia.\nhttp://en.wikipedia.org/wiki/Chikungunya\n\nThe best treatment for Chikunguniya is drinking 6-8 glasses of distilled water each day a pinch of sea salt. This will hydrate and mineralize the body giving it the best ability to flush toxins a relieve body aches. \n\nI would also take mega doses of Vitamin C. \n\nI would also drink unsweetened fruit juice or vegetable juice with\na teaspoon of raw apple cider vinegar with a dash of cayenne, turmeric,  and cinamon to provide good flora and PH balance to speed up recovery.  \n\nIf you have access to the herb Boneset herb also called comfrey, knitbone, Snake Root, and blackwort, it is a very good anti-inflamatory.\nhttp://curezone.com/blogs/f.asp?f=687&t=28860\n\nMSM or Glucosamine are supplements that could help with body aches.
287035
The ZOMBIE is right,IT IS INTERESTING;) \nCurrent International Research suggests that the virus is killed within seconds of reaching 70 degree C. Avoid food that conatins raw meat or eggs!any properly cooked chicken or egg poses no threat!\n\nBon Apetit
221012
dengue,malaria,laberia,kala azar,bird flu,
734972
The rabies death theory was proposed by Dr. R. Michael Benitez, and is based upon the fact that Poe's symptoms before death are similar to those displayed in a classic case of rabies. Cats play a prominent part in many of his stories, and it is conjectured that he was accidentally bitten by a rabid pet.\n\nCats, dogs, ferrets, raccoons, skunks, foxes, wolves, coyotes, bears, bats, and horses can become rabid. Squirrels, chipmunks, other rodents (except beavers), and rabbits are very seldom infected. Rabies may also be present in a so-called "paralytic" form, rendering the victim abnormally quiet and withdrawn.\n\nThe virus is usually present in the saliva of a symptomatic rabid animal; the route of infection is nearly always by a bite, and causes the victim to be exceptionally aggressive[citation needed]. Transmission has occurred via an aerosol through mucous membranes; transmission in this form may have happened in people exploring caves populated by rabid bats. Transmission between humans is extremely rare, though it can happen through transplant surgery (see below for recent cases), or even more rarely through bites or kisses.\n\nAfter a typical human infection by bite, the virus directly or indirectly enters the peripheral nervous system. It then travels along the nerves towards the central nervous system. During this phase, the virus cannot be easily detected within the host, and vaccination may still confer cell-mediated immunity to preempt symptomatic rabies. Once the virus reaches the brain, it rapidly causes encephalitis and symptoms appear. It may also inflame the spinal cord producing myelitis.\n\nThe period between infection and the first flu-like symptoms is normally three to twelve weeks, but can be as long as two years. Soon after, the symptoms expand to cerebral dysfunction, anxiety, insomnia, confusion, agitation, abnormal behavior, hallucinations, progressing to delirium. The production of large quantities of saliva and tears coupled with an inability to speak or swallow are typical during the later stages of the disease; this can result in "hydrophobia". Death almost invariably results two to ten days after the first symptoms; the few humans who are known to have survived the disease were all left with severe brain damage, with the recent exception of Jeanna Giese.
799612
it is dengue\nan acute infectious endemic and epidemic disease caused by a virus transmitted by mosquitoes causes severe pain in bones and muscles and swelling reddening and pain in the joints \nat times it causes skin eruption\nincubation 3-5 days \npresents suddenly with high fever of 3-4 days then subsides followed by a second episode\nrecovery is slow\ncomplications rare \nis widespread in tropical and sub-tropical regions\nalso known as breakbone fever or dandy fever
992756
Any mammal can be a carrier of rabies.
923213
be allert of dengue\nuse mortin\nDengue fever and dengue hemorrhagic fever  are acute febrile diseases, found in the tropics, with a geographical spread similar to malaria. Caused by virus serotypes of the genus Flavivirus, family Flaviviridae, each serotype is sufficiently different that there is no cross-protection and epidemics caused by multiple serotypes (hyperendemicity) can occur. Dengue is transmitted to humans by the mosquito Aedes aegypti (rarely Aedes albopictus).\n\nSymptoms\n\nThis infectious disease is manifested by a sudden onset of fever, with severe headache, muscle and joint pains (myalgias and arthralgias — severe pain gives it the name break-bone fever or bonecrusher disease) and rashes; the dengue rash is characteristically bright red petechia and usually appears first on the lower limbs and the chest - in some patients, it spreads to cover most of the body. There may also be gastritis with some combination of associated abdominal pain, nausea, vomiting or diarrhea.\n\nThe classic dengue fever lasts about six to seven days, with a smaller peak of fever at the trailing end of the fever (the so-called "biphasic pattern"). Clinically, the platelet count will drop until the patient's temperature is normal.\n\nCases of DHF also shows higher fever, haemorrhagic phenomena, thrombocytopenia and haemoconcentration. A small proportion of cases leads to dengue shock syndrome (DSS) which has a high mortality rate.\n\nDiagnosis\n\nThe diagnosis of dengue is usually made clinically. The classic picture is high fever with no localising source of infection, a petechial rash with thrombocytopenia and relative leukopenia.\n\nThere exists a WHO definition of dengue haemorrhagic fever that has been in use since 1975; all four criteria must be fulfilled:\n\n1. Fever\n2. Haemorrhagic tendency (positive tourniquet test, spontaneous bruising, bleeding from mucosa, gingiva, injection sites, etc.; vomiting blood, or bloody diarrhea)\n3. Thrombocytopaenia (<100 platelets per mm³ or estimated as less than 3 platelets per high power field)\n4. Evidence of plasma leakage (hematocrit more than 20% higher than expected, or drop in haematocrit of 20% or more from baseline following IV fluid, pleural effusion, ascites, hypoproteinaemia)\n\nDengue shock syndrome is defined as dengue haemorrhagic fever plus:\n\n# Weak rapid pulse,\n# Narrow pulse pressure (less than 20 mm Hg)\n\n# Hypotension for age;\n#Cold, clammy skin and restlessness.\n\nSerology and PCR (polymerase chain reaction) studies are available to confirm the diagnosis of dengue if clinically indicated.\n\nTreatment\n\nThe mainstay of treatment is supportive therapy. The patient is encouraged to keep up oral intake, especially of oral fluids. If the patient is unable to maintain oral intake, supplementation with intravenous fluids may be necessary to prevent dehydration and significant hemoconcentration. A platelet transfusion is rarely indicated if the platelet level drops significantly or if there is significant bleeding. But the transfusion is recommendable on platelet count falling below 20,000 without hemorrhage / bleeding or approx 50,000 with hemorrhage/bleeding. Internal bleeding indicated by dark color of stools, other bleedings indicated at surface as red rashes all over or most of the body parts.
208517
The virus comes from the Filoviridae family, of which Marburg virus is also a member. The true vector, an animal or plant that can carry the virus without experiencing symptoms of infection, is not currently known. The Ebola virus is named after the Ebola River in the Democratic Republic of the Congo (formerly Zaire), near the first epidemics.\n\nIt is traditional to name viral species (strains, subtypes) after the locations where they were first discovered. Two species were identified in 1976: Zaire ebolavirus (ZEBOV) and Sudan ebolavirus (SEBOV) with case fatality rates of 83% and 54% respectively. A third species, Reston ebolavirus (REBOV), was discovered in November 1989 in a group of monkeys (Macaca fascicularis) imported from the Philippines to the Hazleton Primate Quarantine Unit in Reston, Virginia (USA).\n\nFurther outbreaks have occurred in Zaire/Democratic Republic of the Congo (1995 and 2003), Gabon (1994, 1995 and 1996), Uganda (2000), Angola (2005) and Sudan again (2004). A new species was identified from a single human case in Côte d'Ivoire in 1994, Ivory Coast ebolavirus (ICEBOV). In 2003, 120 people died in Etoumbi, Republic of Congo, which has been the site of four recent outbreaks, including one in May 2005.\n\nOf the approximate 1,500 identified Ebola cases worldwide, over 80% of the patients have died. Despite considerable effort by the World Health Organization, no animal or arthropod reservoir capable of sustaining the virus between outbreaks has been identified, although a role for fruit or insectivorous bats is often postulated. BBC News reported that researchers writing in the December 1, 2005, issue of Nature had identified evidence of symptomless Ebola infection in three species of fruit bats from the Democratic Republic of Congo and Gabon.\n\n[edit]\nBlack Death?\nTher are a group of Scientists who believe that the English Black Death was in fact a large Epedemic of Ebola. This remains to be confirmed.\n\n[edit]\nZaire ebolavirus\nZaire ebolavirus, is a non-segmented, negative stranded virus and is one of the first-discovered Ebola virus species. It is also the most deadly with up to a 90% mortality rate in some epidemics. There have been more outbreaks of Zaire ebolavirus than any other strain. The first outbreak took place on August 26, 1976 in Yambuku, a town in northern Zaire (now the Democratic Republic of the Congo). The first recorded case (not the index case) was a Mabalo Lokela, a 44 year old school teacher just returning from a trip around Northern Zaire, who was examined at a hospital run by Belgian nuns. His high fever was diagnosed as possible malaria, therefore he was given a quinine shot. Lokela returned to the hospital every day. A week later, his symptoms included uncontrolled vomiting, severe diarrhea, headache, dizziness, and trouble breathing. Later, the bleeding began from his nose, mouth, and rectum. Mabalo Lokela died on September 8, 1976, roughly 14 days after the onset of symptoms.\n\nSoon after, more patients arrived with varying but similar symptoms: fever, headache, muscle/joint aches, fatigue, nausea, dizziness etc. which often progressed to bloody diarrhea, severe vomiting, and bleeding from the nose, mouth, and rectum. The initial transmission was believed to be due to reuse of the needle for Lokelas's injection without sterilization (a common practice in many countries). Subsequent transmission was also due to care of the sick patients without barrier nursing and traditional burial preparations, which involves washing and GI tract cleansing.\n\nA similar case of hospital transmission occurred in southern Sudan, after the death of a nightclub owner in Nzara who could afford to go to the fancier hospital located in Maridi. Unfortunately, the nurses there also did not properly sterilize their needles, and the hospital, like the one in Yambuku, became a breeding ground for new Ebola cases (Draper 30–31). Several epidemics of Zaire ebolavirus and Sudan ebolavir
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They have some differences.\nYellow fever (also called yellow jack, black vomit, or sometimes American Plague) is an acute viral disease. It is still an important cause of hemorrhagic illness in several African and South American countries despite existence of an effective vaccine. In the past it was a source of several devastating epidemics. As of 2005, the World Health Organization (WHO) estimates that yellow fever causes 200,000 illnesses and 30,000 deaths every year in unvaccinated populations.\n\nThe disease is caused by an arbovirus of the family Flaviviridae, and is one of the smallest RNA viruses isolated by man.\n\nMalaria, derived from mala aria (Italian for "bad air") and formerly called ague or marsh fever in English, is an infectious disease which causes about 350-500 million infections with humans and approximately 1.3 million deaths annually, mainly in the tropics. Sub-Saharan Africa accounts for 85% of these fatalities.[1]\n\nMalaria is caused by the protozoan parasites of the genus Plasmodium (of the phylum Apicomplexa), and the transmission vector for human malarial parasite is the female Anopheles mosquito. The P. falciparum variety of the parasite accounts for 80% of cases and 90% of deaths. Children under the age of five and pregnant women are the most vulnerable to the severe forms of malaria
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Dengue fever (IPA: ['deŋgeɪ]) and dengue hemorrhagic fever (DHF) are acute febrile diseases, found in the tropics, with a geographical spread similar to malaria. Caused by one of four closely related virus serotypes of the genus Flavivirus, family Flaviviridae, each serotype is sufficiently different that there is no cross-protection and epidemics caused by multiple serotypes (hyperendemicity) can occur. Dengue is transmitted to humans by the mosquito Aedes aegypti (rarely Aedes albopictus).\n\nFind out more about it here :\nhttp://en.wikipedia.org/wiki/Dengue_fever
549641
Bats can get the rabies virus (by biting or being bitten by an infected animal), but the incidence of bats and rabies is very low; usually only about one half of one percent of bats tested, test positive for the virus.
502830
they probably have rabies
640431
bordetella pertusis\ntrypanosoma gambiensis\nneiserria gonnorheae\ntreponema pallidum\nclostridium botulinum\nplasmodium vivax\nsalmonela thippi\ndiplococcus pneumonia
721394
Mosquito or frog eggs