dose oxiecottin have morphene in it?
549113
No.  I've been told that they have similar effects, but morphine is a bit stronger.  The main difference is that morphine is a natural opiate and Oxycontin is synthetically manufactured.
424744
I really only know of one that comes to my mind. Thats grapefruit. If you are on meds it would say that on the label.
10467
As the sodium content in this drug is not considered a 'medical ingrediant' one can assume it is very low.  Here's a direct copy:\n\nSuppliedSupplied: Caplets (Gel): Each oval-shaped, gelatin-coated tablet contains: ibuprofen 200 mg. Nonmedicinal ingredients, cellulose, cornstarch, croscarmellose sodium, FD&C Red No. 40, FD&C Yellow No. 6, gelatin, glycerin, iron oxides, lecithin, pharmaceutical glaze, pregelatinized starch, propylgallate, silicon dioxide, simethicone, sodium lauryl sulfate, stearic acid, titanium dioxide and triacetin. Bottles of 16, 32 and 72. \n\nChildren's Suspension: Each 5 mL of fruit-flavored or grape-flavored suspension contains: ibuprofen 100 mg. Nonmedicinal ingredients: cellulose gum, citric acid, disodium EDTA, FD&C Blue No. 1 (grape-flavored only), FD&C Red No. 40, flavors, glycerin, microcrystalline cellulose, polysorbate 80, sodium benzoate, sorbitol, sucrose, water and xanthan gum. Bottles of 100 mL. \n\nTablets/Caplets (Sugar-coated): Each brown, sugar-coated tablet/caplet contains: ibuprofen 200 mg. Nonmedicinal ingredients: acetylated monoglyceride, beeswax, carnauba wax, cellulose, cornstarch, croscarmellose sodium, ethoxyethanol, iron oxides, lecithin, parabens, pharmaceutical glaze, pharmaceutical shellac, povidone, pregelatinized starch, silicon dioxide, simethicone, sodium benzoate, sodium lauryl sulfate, stearic acid, sucrose and titanium dioxide. Blister packs of 8 tablets. Bottles of 24, 50 and 100 tablets or caplets. Easy-to-open bottles of 150 tablets.
364234
The opthalmic solution's structure can be found here.\n\nhttp://www.pharma.us.novartis.com/product/pi/pdf/voltaren_ophthalmic.pdf#search='Diclofenac%20Sodium%20structural%20formula'\n\nI've worked mainly with the tablets, and the structure for it is as follows:http://www.fda.gov/cder/foi/label/2001/20254s2lbl.pdf#search='Diclofenac%20Sodium%20tablets%20structural%20formula'  \n\nwhere R is either Na or K depending on whether you're dealing with Diclofenac sodium or potassium (I test both).
443548
No; it doesn't. Both medicines are in the same class of drugs, called NSAIDS (Non Steroidal Anti Inflammatory Drugs). You can EITHER be on 1 or the other, but not on both at the same time.\nSo, no; naproxyn does not contain ibuprofen.
833843
Ergometrine alkaloid will show Violet glow in U.V light
50804
the spines don't contain toxins at all-the reaction was probably from the spines themselves-there are also small fine spines in small clusters on some prickle pears (opuntia) that look like hair called 'glochids' that are a real pain when you get them in your skin-it's hard to see at first that they are there because they are so fine and just brushing your hand against them can over your hand in them-i usually use packing tape to pull them out. for the bigger needles try pulling them out gently to not break them and using an antiseptic is good too because infections are possible from bacteria that might have been on the spine although i've never gotten an infections from the many spines that have pricked me:)
780474
Black Cohosh and Soy Isoflavinoids. Will return you to normal, at Wal Mart.
724785
simple answer, no.\n\nthe bugs are one small symptom of M. another is 'brain fog', lots of confusion, and the inability to come to conclusions. The 'bugs' are accompanied with lesions & skin excretions in the form of small balls and filaments .\n\nI am sure diet, sleep, walking will help... but this could be more serious and may require a doctor's visit.\n\nI am not a doctor, but a Morgellons survivor!\n\nvisit http://morgellons.org and take the test! I don't think her symptoms match up.\n\ngood luck & good health.
846811
sounds like cysitis to me. go to a GP it can be damagin, you can get stuff from the chemist for it, try it you never know, personally i would see a GP to confirm.
469369
James L. Schaller, MD, MAR, PA, DABPN, DABFM\n\nClinical and Research Psychiatry and Medicine\nAdult Psychiatry and Psychotherapy Services\nSubspecialty Child & Adolescent Psychiatry\nHormonal & Nutraceutical Consultation\nAnti-Aging & Obesity Consultation\n\nwww.personalconsult.com\nOchratoxins and Ochratoxicosis\nBy Jack Dwayne Thrasher, Ph.D.\ntoxicology@drthrasher.org\n\nIntroduction\n\nOchratoxins are metabolites of Aspergillus, Fusarium, and Penicillium species. The most commonly implicated species are A. ochraceus and ostianus, P. viridicatum, griseofulvum and possibly solitum.1 Recent evidence shows that these mycotoxins are present in a variety of foods (cereals, pork, poultry, coffee, beer, wine, grape juice and milk. Analyses of these food products demonstrated that ochratoxins are also produced by P. vercucosum and A. niger and carbonarius.2,3\n\nThere are three generally recognized ochratoxins, designated A, B and C. Ochratoxin A (OTA) is chlorinated and is the most toxic, followed by OTB and OTC. Chemically they are described as 3,4-dihydro-methylisocoumarin derivative linked with an amide bond to the amino group of L-b-phenylalanine (Fig. 1).4\n\nFigure 1. Chemical Structure of Ochratoxin A (OTA); Adapted from Hussein et al, 2001, ref. 4\n\nThe role and risk assessment of OTA in animal and human disease has been reviewed. The tolerable dosage in humans was estimated at 0.2 to 4.2 ng/kg body weight based upon NTP carcinogenicity study in rats. OTA is mutagenic, immunosuppressive and teratogenic in several species of animals. Its target organs are the kidneys (nephropathy) and the developing nervous system.5,6 Following intravenous administration OTA is eliminated with a half-life from body in Vervet monkeys in 19-21 days.7 There is no reason to suspect that the elimination half-life would be significantly different in humans. OTA has been detected in human blood and milk. Other than nephropathy and urinary tumors, the evidence for human toxicology is scant. The toxicology of OTA will be briefly reviewed.\n\nCarcinogenesis\n\nThere is inadequate evidence that OTA is carcinogenic in humans. There is sufficient evidence in experimental animals. Overall OTA is possibly carcinogenic to humans and is a Group 2B carcinogen.\n\nResearch Animals: Oral administration of OTA produced tubular cell carcinomas of the kidneys in male and female rats, and fibroadenomas of the mammary gland in the females.1\n\nAdministration of OTA in the diet of ddy mice produced tumors of kidneys: solid renal tumors and cystic adenomas. Also, hepatic-tumors and hyperplastic liver nodules were found and unspecified lung tumors. In addition, solid renal-cell tumors occurred in ddd mice fed OTA in their diet.1\n\nMale and Female Fischer rats given oral doses of OTA had does related increased in kidney tumors: renal-cell adenomas, and renal-cell adeno-carcinomas. Metastasis of the renal cell tumors occurred in 17 males and one female.1\n\nHumans: Balkan endemic neuropathy (BEN) associated with OTA occurs in Europe (Bulgaria, Croatia, Turkey, Egypt, and Yugoslovia) where OTA is relatively high in the diet. Individuals with BEN were surveyed for the presence of urinary tract tumors. The incidence of tumors in the urinary system was elevated in both men and women. Furthermore, the observations suggested that individuals with urinary tract tumors had elevated levels of OTA in the blood and urine. Approximately one-third of patients dying from BEN have papillomas and/or carcinomas of the renal pelvis, ureter or bladder.1,8 -12\n\nRecently, it has been suggested that OTA can cause testicular cancer in humans.13 The hypothesis that consumption of foods contaminated with OTA causes testicular cancer was tested. The incidence of rates of testicular cancer in 20 countries were significantly correlated with the per-capita consumption of coffee and pigmeat. Scwhartz concluded: "Thus, OTA is a biologically plausible cause of testicular cancer. Future epide
326763
Cataracts & Vision - Carnosine and N-Acetylcarnosine Eye Drops\nCategories\nHealth through Nutrition\n(http://www.newmediaexplorer.org/chris/2006/03/16/cataracts_vision_carnosine_and_nacetylcarnosine_eye_drops.htm)\nPractical Health\n\nFurther to Cataract - MSM eye drop solution the following provides more information. Although it too discusses some animal data more importantly it also includes some good human clinical data. \n\nEarly cataract is also am indication of calcium overload and sometimes IV EDTA chelation can also reverse it.\n\nI can't emphasis the importance of non processed and low carbohydrate diets for all health conditions...,\n\nSee also: \n\nAvoiding Visual Degeneration\n\nAspartame and Vision Degradation\n \nChris Gupta\n\n---------------------\nCarnosine and N-Acetylcarnosine Eye Drops (original is here)\n\nNew Hope for Cataracts \nWard Dean, MD \n\nCataracts are the leading cause of blindness, accounting for about 42 percent of all cases of blindness worldwide (affecting about 17 million people). Twenty-eight thousand new cases are reported everyday. About 20 percent of all people over 60 have at least the beginning of a cataract in one or both eyes, and that figure rises to 80 percent for people over 75. \n\nThe most common type of cataract ­ a nuclear cataract ­ is characterized by a cloudy haze inside the lens (Fig. 1). This haze is the physical manifestation of a random clumping together of the once beautifully ordered arrangement of lens proteins called crystallins. As the cataract develops in size and density, it reduces the amount of light that passes through the lens and scatters the light that does get through. Thus, instead of all the light rays being focused precisely to a point on the retina, forming a sharp, clear image of what we are seeing, many of the rays are spread out across the retina, forming a fuzzy image. Colors may be dulled or distorted, and there may be an annoying halo of light around bright objects, causing a glare effect.\n\nCauses of Cataracts \nCauses of cataracts include cumulative ultraviolet radiation damage from sun exposure, heredity, poor nutrition, smoking, high blood pressure, kidney disease, diabetes, and the long-term use of corticosteroids (the last two are major risk factors for cataracts). Oxidative free radicals produced by the above-listed causes are thought to damage vital biomolecules, including lipids and proteins, resulting in the clumping together of these proteins. The antidote to free radicals, of course, is antioxidants, such as glutathione, lipoic acid, and vitamins C and E. Consequently, many scientists believe that abundant consumption of antioxidants may delay the onset of cataracts. \n\nNutrients for Vision \nGood nutrition is a key element of effective prevention for most age-related diseases, and cataracts are no exception. A number of nutrients can benefit our eyes, and may help prevent diseases such as cataracts, glaucoma, and macular degeneration. These nutrients include the tripeptide glutathione (the most abundant and important antioxidant in the human body, critical for protecting the lens from free radical damage); vitamins A and C; vitamin E and some of the B vitamins; various bioflavonoids (especially quercetin and hesperidin) and carotenoids (especially lutein and zeaxanthin); the amino acids taurine, N-acetylcysteine (a precursor of glutathione), and acetyl L-carnitine; the hormone melatonin; the alkaloid vinpocetine; the herbs bilberry, ginkgo, and garlic; the minerals zinc and selenium; and, last but certainly not least, the saturated fatty acid, lipoic acid (“the antioxidant’s antioxidant”), which plays a central role in maintaining the body’s antioxidant network.[1] \n\nCarnosine for Vision \nCarnosine ­ a dipeptide consisting of two amino acids (alanine and histidine) connected to each other by a chemical bond called the peptide bond ­ is one of the most exciting anti-aging nutrients that has recently become widel
390158
here is a good website\n\nhttp://www.oxycontininfocenter.com/\n\nhope it helps!
1154495
I use products by Oxyfresh -- LifeShots.  It contains Russian Adaptogen and Activin® ,Opti-Berry® ,Pine Bark Extract,Chinese Wolf Berry, Pomegranate, Green Tea Extract and Noni Fruit .
1116802
Henna is a dye, the source of which is the shrub lawsonia\nalba. The plant is grown extensively in the Middle East and\nAfrica.7 When the dried leaves are soaked in water and applied\nto the hair, skin, or nails, an auburn to red color develops,\nhence, its worldwide use as a cosmetic agent.7 In some\ncountries, ceremonial and social events, including weddings\nand circumcisions, are celebrated by the application of\nhenna to the skin to create a variety of designs and patterns.\nAn important chemical ingredient of henna is lawsone\n(2-hydroxy-1,4 naphthoquinone), constituting about 1% by\nweight of the crushed leaves. The structure and redox potential\nof lawsone is similar to that of one of the naphthalene\nmetabolites, 1,4 naphthoquinone, a potent oxidant of\nG6PD-deficient red cells. Because of these similarities, together\nwith the knowledge that percutaneous absorption of\nnaphthalane may hemolyze G6PD-deficient red cells, studies\nwere designed to determine whether lawsone also may cause\noxidant injury to red cells.7 These in vitro observations indicate\nthat lawsone, a chemical constituent of henna, is capable of inducing oxidative injury to G6PD-normal red\ncells, and even more so to G6PD (A-)red cells.\n\nLawsone was found to\ncause hemolysis, in a dose-dependent manner, as reflected by\ndecreased blood packed cell volumes and hemoglobin levels\nand by histopathological changes in spleen, liver and\nkidney.4 Although whole body is rarely stained with henna,\nour patient was hennaed throughout the whole body.\nHenna is not only used for cosmetics, but also for medication\nas well. In particularly rural areas in Turkey, local henna\nis applied to the hands and hair for cosmetics in some traditional\nceremonies.\nFor many years, henna has been used for the treatment of\nskin disorders, many of which may be fungal in origin, because\nof the presumed anti-fungal properties of henna, tog\ne t h e r w i t h i t s s t r u c t u r a l s i m i l a r i t y t o 5 -\nhydroxynaphthoquinone, a potent fungicide.7\nSeborrheic dermatitis and fungal infestations are other\nreasons why henna is used topically on lesions in some\npeople. In this case, henna was used for treatment of psoriatic\nlesions. Anti-inflammatory, antipyretic, and analgesic effects\nof henna in rats are reported.8 In some trials, henna is considered\nto have low allergic potential and rarely causes contact\ndermatitis.2,9 A case of a hairdresser who developed an immediate-\ntype hypersensitivity with urticaria, rhinitis, and bronchial\nasthma on exposure to henna is reported by Majoie et\nal.10\nBesides hemolysis, nephrotoxicity is another effect of\nhenna, which is clinically presented with renal enlargement,\nelevated plasma levels of urea and creatinine, and tubuler\nnecrosis.3-5 In this patient, there was no nephrotoxic effect of\nhenna; urea and creatinine were in normal range.\nWe have reported this case because when the literature is\nsearched thoroughly, acute hemolysis after applying henna\nto the whole body is a very rare condition. A local health\neducation program should be established to prevent the use\nof henna dye in G6PD enzyme deficient patients.
967989
I remember a girl in the news who ended up inhaling a piece of the X'nas tree and ended up getting a horrid asthma attack.  For years she lived with it until she was operated on about 10 years later.  After extracting the snall twig the leaf parts still had chlorophyll in it.
497593
Test it for esteroids