what is the full form of CSH in Microbiology? eg: CSH 123,E. coli, a bacterial strain.?
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Cell surface hydrophobicity
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I'm not sure I understand the question, but I'll try anyway.  :)  If you are trying to determine IF you have both Pseudomonas and Klebsiella on in your culture, you can streak a MacConkey agar plate with the culture.  Pseudomonas cannot ferment lactose, so it will grow on the plate but remain colorless.  On the other hand, Klesiella can ferment lactose, so it will also grow on the plate but turn bright pink.  That's probably the easiest way to distinguish the two from a mixed culture.\n\nAs for the "local disinfectant" part of the question...are you trying to pull out these bacteria and then do disinfectant susceptibility testing on them?  If so, you can streak out the plate as mentioned above, then subculture each so you can get a pure culture.  Then, you do disinfectant tests on the bacteria separately by the disk diffusion method, etc.\n\nI hope that helps some!
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bacillus, clostridium, desulfotomaculum, sporolactobacilum
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You can find all the info here:\nhttp://www.cbwinfo.com/Biological/Pathogens/OMSKV.html
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To try and answer the question, rather than tell you all about bacterial cell walls again... I think that yes, the wall is an integral part of the bacterial cell membrane. If it was a generic question on the "functions of carbohydrates in cell membranes", then I'd expect an overview of how different carbohydrates are used in different ways, in different cell membranes, including the bacterial ones.\nIt's a bit unfair of your teacher to fail everyone, however - if no one mentioned it, it sounds as if the question wasn't asked clearly. \nIs anyone in the class thinking of challenging him/her on it? Good luck!
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it's called a gram stain.  if the microbe stains, then it is said to be gram positive... if it doesn't stain (or stains poorly), then it's gram negative.\n\nyou have to use all the stains to complete the gram staining process
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E. coli is the main bacterial found in mammal intestines. Yes you've got them too. Most strains of E. Coli are benign but some cause disease like Montezuma's revenge which causes profuse diarrhea. \n\nThere are useful E. Coli. Because it is so easy to grow it is used in science experiments and used to manufacture DNA.
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Hi there, \n\nMany types of bacteria undergo cell division by a procedure called 'Binary Fission'\nYeast does not undergo cell division, however, it reproduces through 'Budding'\n\nHope this helps. If not, I'd be glad to assist you further. Bye!!\n\nrule wid style n luv@vinny
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here are lists and pictures\n\nhttp://www.buddycom.com/bacteria/gpc.html
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Staphylococcus aureus . Staphylococcus is a very well known genus of bacteria. Colonies are "gold," or yellow on sheep blood agar solid media, hence the name. A common pathogen, boils, acne, wound infections, food poisoning are among a host of conditions caused by this organism. The organism is both pathogenic and invasive. It produces leukotoxin which can kill white blood cells and a wide variety of other toxins. S. aureus is quite pyogenic and in decades past was named Staphylococcus pyogenes, however that specific name is currently applied to one GPC, Streptococcus pyogenes.  \n \nIncreasingly, and especially in hospital, strains of both S. aureus and S. epidermidis have become resistant to the antibiotic DOC, methicillin. Such strains have been labelled Staph MR. Other clinically significant species include, S. haemolyticus, S. hominis and S. saprophyticus. \n\nStaphylococcus sp. (Coagulase-negative) Staphylococcus epidermidis, appears as white colonies on sheep blood agar plates. While both S aureus and S epidermidis are normal inhabitants of the skin, epidermidis had been considered the lesser of the two in virulence. Most S aureus strains test positive for coagulase, clot formation in tube of citrated rabbit plasma. The other species test negative. "Infections by S. epidermidis, S. haemolyticus, and S. hominis, are associated with intravascular devices (prosthetic heart valves and intra-arterial or intravenous lines) and shunts. Also quite common are infections of prosthetic joints, wound infections, osteomyelitis associated with foreign bodies, and endocarditis." Dr GE Kaiser\ncat.cc.md.us/courses/bio141/labmanua/lab15/l15index.html  \n \nMicrococcus sp. is a contaminant virtually every time. One definite exception is when it arrives in hospital accreditation test samples in which it is a "freebie." So it deserves mention. If you have ever looked at it under the microscope, you might wonder about the Micro- part of the name, since on gram stain, the cocci can be quite large. Pairs and clusters are seen. Catalase positive. Growth on mannitol agar. What else. "Three common species of Micrococcus are M. luteus, M. roseus, and M. varians." \nMicrococcus information, medic.med.uth.tmc.edu/path/00001448.htm\nView a gram stain, such as it is, M. luteus, buckman.com/eng/micro101/2256.htm \n \nFamily Streptococcaceae \n"Streptococcus, plural streptococci, any member of a genus (Streptococcus) of spheroidal bacteria in the family Streptococcaceae. The term streptococcus ("twisted berry") refers to the bacteria's characteristic grouping in chains resembling a string of beads." "Streptococci can also be classified by the type of carbohydrate contained in the cell wall, a system called the Lancefield classification." Encyclopedia Britannica Agglutination and immunofluorescent antibody microscopic methods can quickly identify Lancefield groups. \nStreptococcus pyogenes \nStreptococcus pyogenes electron micrographs, Rockefeller University, A downloadable set of eleven electron micrographs of Streptococcus pyogenes of exceptional quality.Click link or thumbnail: rockefeller.edu/vaf/ems.htm \n\n\nLancefield Group A, S. pyogenes species of streptococci cause rheumatic fever, scarlet fever, erysipelas, strep throat, tonsillitis, and other upper respiratory infections. In case you are wondering which streptococcal species is the worst, Streptococcus pyogenes, Lancefield group A is the bad boy in the Strep family. It is so important that physicians don't want any silly mistakes with names. "Pyogenes? What's that?" Physicians are programmed to react to the term "Strep group A."\nA recently sensationalized strain of an especially invasive Lancefield group A Streptococcus carries the terms "pyo-," pus. "-gen-," forming. "necrotizing fascitis," flesh eating, to a very rare extreme. Once established under the skin,
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http://www.sbuniv.edu/~ggray/CHE3345/chp14.html\nhttp://web.odu.edu/sci/tisenhou/chem321/chap12/index.htm\n\nbest luck
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Type B refers to the bacterial strain haing  a specific type of long sugar chains that cover the bacterial cell forming the capsule. U must be aware of the fact that any bacteria a covered by a capsule made of sugar/polysachs. some bacterial strains are classified based on teh type of sugar chains they bear on their surface (which may act as antigen to be recognized by host immune system).
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It depends on the bacterium. Actually there are halophiles which like very high salt concentrations.\nIt also depends on the strain. So there are strains of E.coli which stop growing at around 0.6 M NaCl and others at 1.2 M, though it also depends on the contribution of the rest of the solutes to the osmolality of the medium.
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Heres everything you need to knoe about Ecoli\nhttp://en.wikipedia.org/wiki/E_coli
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Just go straight to Wikipedia to get the answer:\nKingdom: Bacteria\nPhylum: Firmicutes\nClass: Bacilli\nOrder: Lactobacillales\nFamily: Streptococcaceae\nGenus: Streptococcus\nSpecies: S. pneumoniae\nhttp://en.wikipedia.org/wiki/Streptococcus_pneumoniae
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A hfr cell (also called hfr strain) is a bacterium with a conjugative plasmid (often F) integrated into its genomic DNA. Hfr is the abbreviation for high frequency recombination, which was first characterized by Luca Cavalli-Sforza. Unlike a normal F+ cell, hfr strains will, upon conjugation with a F- cell, attempt to transfer their entire DNA through the mating bridges (called pili) to the F- cell. This occurs because the F factor has integrated itself via an insertion point in the bacterial chromosome. Due to the F factor's inherent nature to transfer itself over the pilus during bacteria conjugation, the rest of the bacterial genome is dragged along with it, thus making such cells very useful and interesting in terms of studying gene linkage and recombination.\n\nA structure as fragile as a mating bridge will, however, likely break, and so the transfer is rarely complete. Thus, the F- cell uses only part of the genomic DNA of the Hfr cell for recombination. Though there is some debate on the issue, the pili themselves do not seem to be the structures through which the actual exchange of DNA takes place; rather, some proteins seem to open a channel between the bacteria.\n\nIt should also be noted that the bacterial genome's rate of transfer through the pilus is constant; this allows molecular biologists and geneticists to use Hfr strain of bacteria (often E. Coli) to study genetic linkage and map the chromosome. The procedure commonly used for this is called interrupted mating.
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All organisms use the same four subunits in DNA.  ATCG  There are some bacteria I believe that use one other subunit. Most organisms have RNA because it is how the body decodes DNA into proteins.  In RNA there are also four units those are AUCG