what is anteroseptal myocardial infraction?
541358
well,myocardial infarction is a serious, sudden heart condition usually characterized by varying degrees of chest pain or discomfort, weakness, sweating, nausea, vomiting, and arrhythmias,sometimes causing loss of consciousness,it occurs when the blood supply to a part of the heart is interrupted,causing death and scarring of the local heart tissue,since the area affected may be large or small,the severity of heart attacks vary,but they are often a life-threatening medical emergency which demand both immediate attention and activation of the emergency medical services.\nso, anteroseptal myocardial infarction  is a type of myocardial infarction (as classified according to the site)in which the infarcted area includes the anterior wall of the heart & the septum (interventricular septum).
1135656
Left Anterior Descending Artery\n\nCause many times people get heartattacks due to blockage in this artery, and it happens both fast and unexpectedly.
1012830
Yes you should find out the cause of these pains , then you can decide if you need to worry. If you dont find out the cause then I am going to worry. I have had several sucessful heart attacks, Angioplasty , and quadrupal Bipass Surgary . I am now 64 years old , my first heart attack was when I was 45 but I know many people who have had them when they were much younger. You should know that one of every three people who have heart attacks have little or no warning except sudden death. Dont worry ...Just do somthing!!!
281406
The cause is of paramount concern.\n\nIf new, and from acute coronary disease, then there are great treatment options.  Stenting at your grandfather's age would be a solid option, if multiple vessel disease, may require coronary artery bypass grafting (cabg, we say cabbage).\n\nIf from prolonged hypertension... viral disease... prolonged alcohol abuse... others.. then his heart muscle may be unrecoverable, and he's in bad shape.  Medications to reduce symptoms and increase function work, but ultimately limited by the amount of living/functioning tissue, and more complex things like exactly "how" that tissue is working.\n\nIf from dead tissue due to prior heart attack, more radical surgeries are possible... you'd need an absolute expert, look to a university setting if this is the case.\n\nIf no surgical options, and there is a vast amount of tissue dead or useless... his mortality is in the months (could be days, hours, is he in the ICU?, sorry to scare you).  Transplant can do amazing things if an organ is available, he's sick enough to get the organ, but well enought to survive....\n\nIf he's walking, and just has a cough, and swelling in the legs; with weight loss, excercise, lots of medications, he can nearly restore his mortality to normal (depending on co-existing conditions).
41298
It's cool;\n\nDystrophic calcification is the mineralization of soft tissue without a systemic mineral imbalance. This occurs as a reaction to tissue damage, including as a consequence of medical device implantation.
713697
Endomyocardial fibrosis (EMF) is an idiopathic disorder of the tropical and subtropical regions of the world that is characterized by the development of restrictive cardiomyopathy. Restrictive cardiomyopathy is the least common form and occurs when the middle layer of the heart cavity wall - the myocardium - is excessively rigid, impairing the filling of the ventricles with blood between heartbeats. \nPlease see Google search for more details on Endomyocardial fibrosis.
536839
Your vocab is a LITTLE confusing - there is no such thing as "pulmonary ventricular fibrillation" - just 'ventricular fibrillation'. This is the most serious cardiac arrythmia, and is rapidly fatal unless defibrillated (shocking the chest).\nThe vast majority of coronary angioplasty and stEnting (drug-eluting stents, or bare-metal stents) cases are performed without any occurrence of arrythmias. Especially if the initial blockage was only 80%, ie it was likely performed as an elective procedure, not as an emergency after an acute MI (heart attack).\n\nSo, in short, this is NOT normal.
293214
This answer really depends on your age, sex and risk factors.  If you are young <35 its probably nothing serious but could be your thyroid, panic attack or rarely a heart problem.  If you are older, obese, smoke etc it could be a sign of something more serious in the heart and should be further evaluated by a doctor.  Bottom line if you have already been evaluated by a doctor and had a normal EKG its probably nothing but if you are still nervous you can ask for an event monitor.
276970
An abonormal dilatation of the artial septum with bulging of the septum at least half the distance to the left atrial wall. Congenital heart defect
323698
OK... time to make some clarifications. A 64-slice CT scanner is sometimes used as an alternative to more invasive coronary angiography. The fact that your CT scan showed two areas of blockage means you should probably undergo a coronary angiogram to verify these findings and to treat the disease if it is there. You should definitely start taking an aspirin, as this has been shown in studies to reduce the risk of heart attack in patients with known coronary disease. In addition, there are a handful of other drugs that have been shown in very large, well designed scientific trials to reduce your risk of heart attack, heart failure, and death. These include a beta-blocker (one kind of blood pressure medicine), an ACE-inhibitor (another blood pressure medicine), and a cholesterol-lowering drug in the "statin" class. Several alternative treatments have been tried and pushed in the past including chelation therapy, but the truth is that none of these have been shown to reverse coronary artery blockages. In fact, none of the drugs mentioned above have been shown to reliable reverse coronary blockages either. BUT you don't necessarily need these blockages to shrink for the medications to provide a benefit.\n\nNow, I have to say that IT ISN'T TRUE THAT YOUR 90% BLOCKAGE COULD KILL YOU AT ANY MOMENT. Sure, it's POSSIBLE that you could drop dead tomorrow, but that 90% blockage probably grew gradually over the course of a few years so it is unlikely to suddenly close off at any moment. Don't panic! On the other hand, it will probably continue to progress to 95%... 97%... 99%... and eventually lead to such significant blockage that some of your heart muscle will fail to get enough blood flow, even when you are at rest. This can lead to something called a non-ST elevation MI - a heart attack. The more upstream that blockage is, the more heart muscle it supplies and the more life-threatening that heart attack can be.\n\nWith the newer technologies at our disposal, most significant blockages in the coronary arteries can be opened during the angiogram with a small, drug-eluting stent. These have a very good tendency to stay open over many years, thus avoiding the need for surgery in most patients. However, depending on the location of the blockage, some patients may still do better with surgery.\n\nI strongly urge you to see your doctor and get the coronary angiography done. It is not something that you have to do in the next day or two, but it should definitely be taken care of in the next couple of weeks. Believe me, if there were natural medicines that are effective for treating coronary disease, your insurance company would force you to take those before it agreed to shell out $15,000 for a stent.
468617
go to your doctor..\nand is this your sternum or the xyphoid process, at the tip, because if the xyphoid is broke, it could pierce your lungs or heart..so go to the doctor, get an x-ray
480665
Sorry - height has not been demonstrated to be associated with an increased risk of myocardial infarction (or heart attacks).  The Framingham Heart Study is the seminal study from where we get most of our current knowledge about risk factors for heart disease, and height was NOT a significant risk factor.  But some particularly tall individuals may have Marfan syndrome which is associated, as someone else mentioned, with ascending aortic aneurysms.  In Marfan patients, these aneurysms are at high risk of dissecting (which is what happened to John Ritter).  Dissections feel and present like heart attacks in many ways and are often mislabelled as "heart attacks".  Patients will typically complain of severe chest pain.  Dissection can lead to heart attack, tamponade, stroke, rupture, and death.  I think this is probably what you are talking about.
142992
Diseases of the myocardium\nThere has been increasing recognition of a type of heart disease characterized as primary myocardial disease. The cardiomyopathies are diseases involving the myocardium (heart muscle) itself. They are unique in that they are not the result of hypertensive, congenital, valvular, or pericardial diseases and are rarely the result of ischemic heart disease
475089
cardiac muscle or myocardium
1098099
First I would ask for an echocardiogram with & without saline (water bubbles); this will show any & hopefully all holes in your heart. I had it done & they found I was missing a chamber and had holes on the opposite side of my heart which required them to "cath" my heart. They make a small slice in the groin area & go through a main artery in your leg up to your heart. I'm only 5' & 100 lbs & they couldn't knock me out with morphine (I watched the entire procedure & ticked off my cardiologist -lol). My personal opinion would be 1. ask questions, make a list if you have to, 2. check out the surgeons & their history, & 3. if they are as good as they check out to be the procedure will be a cakewalk for you. I was 38 when they found all of this & now I'm 43 and alive to see my baby (23) get married this coming August to a wonderful young lady. My Dad had 2 triple arterial bypasses & he lost a leg in Korea and God bless him, he'll be 79 this July. Yes, I had a good role model but it is normal to get upset when a Dr mentions a prob w/the heart & that you have to face surgery. I now know how a lobster feels having its chest cracked but thorasic & cardiac surgery is so advanced that I can promise you that you'll be fine. Try to keep a positive attitude and have faith; they told me after 3 days that if I could walk up & down a flight of stairs 4 times, I could go home..........I bolted & got my boney butt outta that hospital & back into my own home in no time. My thoughts & prayers are with you (Navy brat here) but above all : keep the faith.\nBeth
853203
In looking into your inquiry, it turns out that a very rare complication can be artery perforation and resulting cardiac tamponade.  I located a study from back in the 90's about this complication.  Here is some background information that will help you gain perspective on the whole matter.\n\nDuring the 3-year study period, a total of 15 patients had cardiac tamponade during or within 36 hours after 6999 coronary interventions, yielding an incidence of 0.21%. No cases of tamponade occurred among 14,927 patients undergoing diagnostic catheterizations during the same period.\n\nThe diagnosis of tamponade was usually confirmed by echocardiography, with the exception of 1 patient who had hypotension and tamponade physiology while in the laboratory. Fluoroscopy revealed immobile heart borders, and pericardiocentesis was performed immediately without prior echocardiography, with rapid restoration of normal blood pressure. The echocardiogram was performed within 30 minutes of onset of hypotension in 9 of 15 cases, and in all, within 2 hours.\n\nThe clinical circumstances leading to emergency echocardiography and therapy varied, but with a common theme of hypotension. The 6 patients who had tamponade in the catheterization laboratory were immediately recognized on the basis of sudden profound hypotension and (in the 4 coronary artery cases) by obvious contrast extravasation. Of the 9 patients with delayed tamponade, 8 had unexplained hypotension. Four of these also had chest pain, 3 had dizziness, and 2 had severe bradycardia. Only 2 patients had asymptomatic hypotension.\n\nTreatment was surgical in 9 (60%) of 15 patients. These patients had various indications. In 1 patient, tamponade was not suspected until the chest was opened for emergency coronary bypass graft surgery. In 5 patients, pericardiocentesis was not successful in controlling the bleeding, whereas in the remaining 3 patients, pericardiocentesis was not attempted.\n\nThe mean time from surgical consultation to arrival in the operating room was 1 hour. Four patients had surgery as the primary treatment, whereas 5 had a percutaneous pericardial drain placed but required surgery for continued active bleeding. Six (40%) patients responded well to conservative treatment with pericardiocentesis alone. In cases of coronary perforation, prolonged balloon inflations, often with the use of perfusion balloons, usually were used to achieve hemostasis or as a temporizing measure before pericardiocentesis.\n\nHere is a case of having cardiac tamponade/pericarditis incidental to the procedure.  Two problems not necessarily related to each other.  Infectious complications of coronary angioplasty and stenting are uncommon. A 70-year-old man is presented who underwent percutaneuous transluminal coronary angioplasty (PTCA) and stenting of an occluded left anterior descending artery. This was complicated by Staphylococcus aureus pericarditis with tamponade. He was successfully treated with a closed drainage and antibiotics. This is the first reported case in the literature that documents purulent pericarditis and tamponade following percutaneous revascularization.\n\nBacterial pericarditis is very unlikely but then your whole experience has been very unlikely.  See the attached link to learn more about pericarditis, which is what you are currently suffering from.  As you will see viruses and inflammatory diseases can also account for this presentation too.  \n\nhttp://www.emedicine.com/med/topic1781.htm\n\nSo now that I have done all of this footwork for you...two questions:  1)  Why couldn't you look this up yourself?  2)  Why didn't you select a best answer on your last question about this matter?
58459
Call and/or go to your doctor immediately. It could just be anxiety or indigestion, but you want to rule out myocardial infarction aka heart attack. Your doc can usually run a couple simple tests. You need to do so before it's too late. Good luck.