is there a certain age that you can get affected by sickle cell anemia? if so, what age?
448941
No, sickle cell is a disease the people are born with.  It only occurs in African Americans or people of African American decent.\n\nHere is a page that makes it easy to understand...\n\nhttp://kidshealth.org/teen/diseases_conditions/blood/sickle_cell_anemia.html
601954
agree with 12-14 g/dL\n\na little lower for adult women, and a little higher for adult men, since they don't menstruate.\n\nlower than 10 is anemic, and some people require blood transfusions when they drop below 9.
1113703
im not aware of any diseases associated with blood types A + B. the only issue is if a mom as a non-compatible blood type with her baby, sometimes that can cause a problem but even that is usually not an issue
1008915
it has been discovered by physicians that 18 is the oldest.so if yuo havent dont despair i started mine when i was 14.
782788
There really is no specific age group for this serious condition. It is easily transmitted by blood or other bodily fluids. There is treatment but no cure. Emergency medical workers fear this as much as HIV.
402162
Thalassemia is the name of a group of genetic blood disorders. To understand how thalassemia affects the human body, you must first understand a little about how blood is made.\n\n Hemoglobin is the oxygen-carrying component of the red blood cells. It consists of two different proteins, an alpha and a beta. If the body doesn't produce enough of either of these two proteins, the red blood cells do not form properly and cannot carry sufficient oxygen. The result is anemia that begins in early childhood and lasts throughout life.\n\nSince thalassemia is not a single disorder but a group of related disorders that affect the human body in similar ways, it is important to understand the differences between the various types of thalassemia.
1149697
it really varies.. there is a couple in my congregation that had a daughter (Abigail) with Spina Bifida and she only lived 3 1/2 years.. i was reading a story online and there was a 82 year old man that had spina bifida, so you really can't put a number on it.. i think it depends on the severity of the condition..
189833
there is no cure for sickle cell anemia
716160
The "sickle cell" gene (technically, allele) is a result of a mutation that modifies the shape of red blood cells. It's what is known as an incompletely recessive trait, meaning that a person with one copy of the sickle cell allele and one copy of the normal allele has mostly normal red blood cells with a few sickled ones. This state has been shown to give the person carrying it resistance to malaria, a common and often fatal disease in tropical regions, particularly Africa. \nThus in Africa, the sickle-cell allele would have been rendered more common by natural selection, since being resistant to malaria was a huge advantage. The children of two people carrying the sickle-cell trait have a 25% chance of receiving both sickle-cell genes from their parents, which is known as sickle-cell anemia. \nIt's not that only people of African descent can have sickle-cell anemia, but through selection the responsible gene was made more common because of malaria resistance. The same would not be the case in areas of the world where malaria was not common; which is why people whose ancestors came from other places are not as likely to have two parents carrying the sickle-cell trait.
1100418
yeah, sickle celled people are resistant to malaraia, so its thought to be a mutation that has helped people adapt to areas high in malaria like africa. so black people have it at a much higher rate than white people
761535
general anaesthesia  presents risk and certain danger to anyone, in particular to those with chronic health problems.  a carrier of the sickle cell trait isn't really a chronic health problem and i would have to agree with your surgeon......he knows best.  but if you are that concerned, get another opionion, say with a hemotologist, specializing in blood disorders.
405880
Have you been tested to see if you are anemic.  I am anemic and when my hemoglobin drops low sometimes I get dark cicles under my eyes that won't go away until my iron levels rise.  Just something to check into
724539
Sickle-cell disease is a general term for a group of genetic disorders caused by sickle hemoglobin (HbS). In many forms of the disease, the red blood cells change shape upon deoxygenation because of polymerization of the abnormal sickle hemoglobin.\n\nHbN and HbS are alleles which are alternative forms of gene. All humans have 2 alleles for every gene (one from each parent).\nNormal healthy people have HbN HbN (HbN=normal hemoglobin).\nCarriers have HbN HbS (HbS=sickle hemogloblin).\nPeople suffering from sickle cell anaemia have HbS HbS. \n\nIn people heterozygous for HbS (carriers), the polymerization problems are minor, hence they don't suffer the effects of sickle cell anaemia. In people homozygous for HbS, the presence of long chain polymers of HbS distort the shape of the red blood cell, from a smooth donut-like shape to ragged and full of spikes, making it fragile and susceptible to breaking within capillaries. Carriers only have symptoms if they are deprived of oxygen (for example, while climbing a mountain) or while severely dehydrated.\n\nThe sufferers of the illness have a reduced life span. It is believed that carriers (sickle cell trait) are relatively resistant to malaria. Since the gene is incompletely recessive, carriers have a few sickle red blood cells at all times, not enough to cause symptoms, but enough to give resistance to malaria. Because of this, heterozygotes (HbS HbN) have a higher fitness than either of the homozygotes (HbN HbN & HbS HbS). This is known as heterozygote advantage. Those with HbN HbN are susceptible to malaria while those with HbS HbS will suffer from sickle cell anaemia.  \n\nThe malaria parasite has a complex life cycle and spends part of it in red blood cells. In a carrier, the presence of the malaria parasite causes the red blood cell to rupture, making the plasmodium unable to reproduce. Further, the polymerization of Hb affects the ability of the parasite to digest Hb in the first place. Therefore, in areas where malaria is a problem, people's chances of survival actually increase if they carry sickle cell anaemia.\n\nDue to the above phenomenon, the illness is still prevalent, especially among people with recent ancestry in malaria-stricken areas, such as Africa, the Mediterranean, India and the Middle East. In fact, sickle-cell anemia is the most common genetic disorder among African Americans; about 1 in every 12 is a carrier.
923777
Sickle cell disease is an inherited disorder of the red blood cells characterized by abnormally shaped red cells. This abnormality can result in painful episodes, serious infections, chronic anemia, and damage to body organs. These effects can, however, vary from person to person depending on the type of sickle cell disease the person has.\n\nSome individuals are relatively healthy and others are hospitalized frequently. But thanks to advancements in early diagnosis and treatment, most children born with this disorder grow up to live relatively healthy and productive lives.\n\nA Closer Look at Sickle Cell Disease\nThe different forms of sickle cell disease are determined by the genes that are inherited from the person's parents.\n\nAn individual has the disease if he or she inherits a sickle cell gene from each parent (hemoglobin SS disease, also called sickle cell anemia).\n\nHe or she can also inherit a sickle cell gene from one parent and a different kind of abnormal gene from the other and end up with a different form of sickle cell disease, such as hemoglobin SC disease and hemoglobin S-thalassemia.\n\nIf an individual inherits only one sickle cell gene and a normal gene from the other parent, then he or she will have the sickle cell trait, but not the disease. A blood test can determine whether you have sickle cell disease or carry the sickle cell trait.\n\nPeople with sickle cell trait don't have sickle cell disease or exhibit any signs of the disorder, but they can pass the gene for the disease to their children. When both parents have the sickle cell trait, there's a 25% chance that a child will have sickle cell disease. But when one parent is carrying the trait and the other actually has the disease, the odds increase to 50% that they will pass the disorder on to their child.\n\nWho Does It Affect?\nIn the United States, hemoglobin SS disease (sickle cell anemia) affects mostly African Americans and some Latino Americans. Some forms of sickle cell disease may occur, although less frequently, in people with different ethnic backgrounds such as those whose ancestors came from Mediterranean countries (including Turkey, Greece, and Italy) or East India.\n\nWhat Causes It?\nA child with sickle cell disease has inherited a defective hemoglobin gene. In a child with sickle cell disease, the hemoglobin can take on an abnormal shape, distorting the shape of the red blood cell. The cells change from a normal round, doughnut shape to the elongated shape of a sickle, or the shape of a "C."\n\nUnlike normal red blood cells, which move easily through small blood vessels, sickle cells are hard and pointed. The sickle shape means that they have a tendency to get stuck in narrow blood vessels and block the flow of blood. This can cause episodes of pain and can also lead to damage of the body organs because they aren't getting enough oxygen.\n\nSickle cells have a shorter-than-normal life span, which leads to anemia (low red blood cell count). A normal red blood cell lives for about 120 days, whereas a sickle cell lives for only 10 to 20 days.\n\nHow Is It Diagnosed?\nUsually, sickle cell disease is diagnosed at birth with a blood test. This can be done in conjunction with other routine newborn screening tests. If your child tests positive on the screening test, a second blood test is usually performed to confirm the diagnosis.\n\nBecause children with sickle cell disease are at an increased risk of infection and other health complications, early diagnosis and treatment is important. Currently, more than 40 states have newborn screening programs for sickle cell disease.\n\nWhat Are the Signs and Symptoms?\nSymptoms of sickle cell disease vary and range from mild to severe, and symptoms may be less severe or different in children who have inherited a sickle cell gene from one parent and a different abnormal hemoglobin gene from the other.\n\nMost children with sickle cell disease have some degree of anemia, an
783184
this may just be your body ..giving blood today is a serious thing and you are nice to want to give but I myself can't so the other problem is the new meds may be to strong for you it is hard to be rid of anemia I have had it all my life you can do everything right it is your body and you have to live with it kara
630617
It depends on  type of leukemia,age of the patient ,previous treatment,relapsee or first time and time of diagnosis.Childhood leukemia is well response to chemotherapy and cure rates are good although relaps are quite common.On regarding G6PD deficiency,it is not a fetal disease.you need to avoid certain drugs that can precipitate haemolysis in these patients.For sickle cell anaemia,it can be treated by blood transfusion.Sickle cell crisis are serious complication .If all these diseases are present in a single person,his life expectancy can be reduced.Among these,leukemia is the worst.The patient will need blood transfusion and chemotherapy.Bone marrow transplantation is the best way to treat this disease but it would be difficult .
878022
If your anemic it means you don't have enough red blood cells. Anemia can be common, especially for women that are menstrating once a month, or serious. I was anemic when I was a kid, but I out gew it with proper nutrition and vitamins.\n\nYou need to eat foods rich in iron or take an iron supplement. Maybe your doctor gave you a prescription. \n\nHere are some other resources for you.\n\nhttp://www.kidshealth.org/parent/medical/heart/anemia.