what are some common names for acid maltase deficiency?
85891
The Maltase Falcon Syndrome
817866
Low sugar level.\nMay be worth having a diabetes check with the doctor, but this can be hormonal.
255363
Beriberi disease is caused by a thiamine (vitamin-B) deficiency. The biologically useful form of thiamine is called TPP or thiamine pyrophosphate (or TDP- thiamine diphosphate), and it is used in biological processes, like the Krebs Cycle and the Pentose Phosphate Pathway. \n\nThiamine pyrophosphate (TPP) is a coenzyme for pyruvate dehydrogenase, α-ketoglutarate dehydrogenase and transketolase. The first two of these enzymes function in the metabolism of carbohydrates, while transketolase functions in the pentose phosphate pathway to synthesize NADPH and the pentose sugars deoxyribose and ribose. TPP is synthesized by the enzyme thiamine pyrophosphokinase, which requires free thiamine, magnesium, and adenosine triphosphate. It is yet unclear exactly how thiamine plays a role in neurological function, but it is found in nerves and in the brain and the lack of it causes neurological disorders.
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AVANDIA, ACTOS,GLUCOPHAGE, GLUCOVANCE,  \nHUMALIN, HUMALOG
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Glucose, Fructose, Maltose
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Malic acid and citric acid.\nThe first one is also found in apples.
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alternate passionate  nativity marinate obstinate fortunate unfortunate vaccinate  resonate abomination combination incriminate procrastinate eliminate ornate donate unattractive nation termnate inseminate  opinionated coordinate signature\n\nwhew...... my brain hurts....... hope you get an "A" and  this was tough for 2 points!
269931
glucose, fructose, galactose are the most common.
499853
Amylase.  It's found in the mouth, as well as stomach and small intestine.
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nena\nneneng\nmiguel\nchayong\nnarciso\ndeogracias\n\nhttp://www.medicaliterature.com/Pag-aaral.shtml
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Impaired tissue perfusion r/t vascular damage.\nRisk for injury r/t decreased sensation.\nRisk for infection r/t impaired tissue perfusion.\nKnowledge deficit r/t diet, checking glucose, and administering insulin injections.\n\n\nGo buy yourself a nursing care plan book.  Those list more nursing diagnosis and interventions for each disease than you can shake a stick at!  Good luck in school.
698394
The classic deficiency syndrome of niacin is called pellagra. Because of niacin's crucial role in the most basic biochemical reactions, every cell in the body can be affected by a deficiency. The three major "target" organs of pellagra are the skin, the gastrointestinal tract, and the nervous system. There is some evidence that a prolonged deficiency of niacin can result in a more or less permanent "dependency," or increased requirement, for the vitamin.\n\nThe best-known symptoms of niacin deficiency, or pellagra, are the "3-Ds": dermatitis, diarrhea, and dementia. The dermatitis in part resembles that of other B vitamin deficiencies, with swollen, bluish tongue, and inflamed mouth and lips. But these are the least of the manifestations on the skin. The skin may become red, blistery, infected, fissured, scaly, or hardened. These lesions usually appear on areas of the body exposed to sunlight or trauma. Elbows and knees, the backs of the neck and hands, and the forearms are common areas afflicted. The mucous membranes of the mouth may become sore and ulcerated and eventually hemorrhage.\n\nThe mucous membranes of the gastrointestinal tract are similarly affected. Digestive secretions are diminished, and burning, gastric discomfort, distention, flatulence, and occasional vomiting give way to severe diarrhea.\n\nA comparable disaster takes place in the nervous system. The first symptoms are mild anxiety, apprehension, fatigue, loss of appetite, skin hypersensitivity, digestive upset, headache, insomnia, tension, and alternating depression and hyperactivity. These progress to irritability, emotional instability, and various psychotic symptoms such as hallucinations, confusion, disorientation, paranoia, delirium, mania, failing vision, hypersensitivity to light, hyper acute sense of smell, dulled sense of taste, and severe depression.\n\nThere is a group of symptoms sometimes found in niacin deficiency distinct from pellagra. Some of the less severe manifestations of dermatitis may be present, along with clouding of consciousness, "cogwheel" rigidity of the extremities, uncontrollable grasping and sucking reflexes, and coma. This syndrome generally appears in hospitalized people who have been maintained on intravenous feeding without vitamin supplementation for long periods of time.\n\nBecause niacin dilates the blood vessels, temporarily lowers blood pressure, and appears to lower blood cholesterol, there is a certain amount of controversy surrounding its use as a treatment in heart disease. The primary problem in heart disease is the blockage of blood vessels. Any factor which widens the blood vessels and restores circulation is of possible benefit. High blood pressure and blood cholesterol also appear to heighten the risk of developing heart disease. So any factor which can lower blood pressure and/or cholesterol levels is a potential aid, too.\n\nVitamin B3 ( Niacin )\nNiacin's basic function is to assist in cellular respiration and in the cell's utilization of all major nutrients. The two coenzymes which require niacin have been identified as necessary to at least forty different biochemical reactions. Without question, niacin is one of the most important factors in keeping us moving toward the optimum health end of the continuum.
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Common Monosaccharides:\n1) Glucose\n2) Fructose\n3) Galactose\n\nCommon Disaccharides:\n1) Lactose (Galactose + Glucose)\n2) Sucrose (Fructose + Glucose\n\nCommon polysaccharides:\n1) Glycogen\n2) Cellulose\n3) Plant starch composed of amylose and amylopectin
190080
Treatment    Return to top \n\nTreatment for this disease concentrates on avoiding low blood sugar. Frequent feedings are needed during the day, especially foods that contain carbohydrates (starches). Neither fruit nor milk sugar can be adequately broken down in this disease, and are best avoided.\n\nOvernight fasting is avoided by continuous feedings (through a tube that goes through the nose into the stomach) of sugars or uncooked cornstarch. The tube can be put in at bedtime and taken out each morning. A medication called allopurinol, which can lower blood uric acid and decrease the risk for gout, is often used.\n\nSynonyms:\nvon Gierkes disease\nGlucose-6-Phosphatase Deficiency Glycogen Storage Disease\nType I Glycogenosis\nHepatorenal Glycogenosis\n\nIn Type I Glycogen Storage Disease, the enzyme glucose-6-phosphatase is defective in the liver, kidney, and small intestine, where it normally functions. However, the metabolic problem is centered in the liver.\n\nPatients with Type I GSD are unable to release glucose from glycogen. They cannot maintain their blood glucose levels and within a few hours after eating develop hypoglycemia (low blood sugar). The low levels of glucose in the blood of these patients often result in chronic hunger, fatigue, and irritability that is especially noticeable in infants. Symptoms of hypoglycemia only begin to appear when the interval between feedings increases and the infant sleeps through the night or when an illness prevents normal feeding routine. If the blood sugar falls to a low enough point, some patients may experience seizures. \n\nSince Type I patients are able to store glucose as glycogen but not release it normally, with time the stored glycogen builds up in the liver. Hormones, lactic acid, triglycerides, and other products are increased in the blood as the body attempts to raise blood sugar. Fats are stored in the liver along with the glycogen, which leads to enlargement of the liver. The liver does its many other functions normally, and there is not usually any evidence of liver failure. The kidneys are also enlarged due to glycogen accumulation. \n\nThe continued presence of low blood sugar eventually leads the body to experience severe biochemical abnormalities and delayed growth and development. Untreated patients may have muscle wasting and a buildup of body fat.\n\nWhen blood studies are done, it is discovered that there is usually a low blood sugar, elevated blood lactic acid, elevated cholesterol and other fats, and elevated uric acid concentration. High blood pressure has also been seen in a number of patients and when this occurs, appropriate treatment must be given.\n\nIn addition to the problems described above, patients with Glycogen Storage Disease Type Ib can develop frequent bacterial and fungal infections, due to abnormal functioning of the white blood cells. They may also develop chronic pancreatitis, chronic inflammatory bowel disease, and Crohn's disease. \n\nThe diagnosis of Type I GSD will always include blood studies, x-rays (including some of the kidneys), measurements, and ultrasound of the liver. Gene-based mutation analysis can be used to diagnose the majority of Type I patients, while liver biopsy analysis may be needed in some cases. \n\nThe treatments of Type I Glycogen Storage Disease are aimed at correcting the biochemical abnormalities and promoting growth and development. Current treatments consist of providing a continuous source of glucose by giving the patient glucose drinks frequently during the day and, in most cases, continuously overnight. Many medical centers have replaced these glucose feedings with uncooked cornstarch doses, mixed in water, Kool-Aid, or formula. \n\nLinks:\nvon Gierke Disease info from National Organization for Rare Disorders            [NORD]\nOMIM GSD Type I on Online Mendelian Inheritance in Man\n     searchable database\nGSD Type I from emedicine\nAmerican Liver Foundation information on Type I\nvon Gierke D
100081
Litmus tests for acidity are probably the most well-known, but there are lots and lots of them. The Encyclopaedia Britannica article below is pretty thorough.\n\nGood luck.
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PLAB  \n... · gpvts · mrcgp · mrcog · mrcp · mrcpath · mrcpch · mrcs · plab · toefl · usmle ... DM - Gastroenterology DM - Neurology DM Endocrinology DM Nephrology DNB Part 1 ...www.rxpgonline.com/plab.html \nDNB Forums  \n... · gpvts · mrcgp · mrcog · mrcp · mrcpath · mrcpch · mrcs · plab · toefl · usmle ... DM - Gastroenterology DM - Neurology DM Endocrinology DM Nephrology DNB Part 1 ...www.rxpgonline.com/forum29.html
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weak organic acid= vinegar\nTitanium Oxide = sun block\nalkaline earth hydroxide= milk of magnesia\nalkali metal= NaCl\npaper chromatography= a penny and iron vinegar mixture\ntransition element= iron or copper\nnonmetal=sugar\nelectrolyte= NaCl\na noneletrolyte solution= sugar and water\noxide  of a element = rust or sun block