Is there any scientific study of the long-term effects of DHE <hallucinogenic like LSD> on the human body?
91136
*Shaking my shaggy skull* As DHE is relatively new, there are no long term studies that I can find so far, a lot of short term ones though. Most studies of that sort are under controled situations for things like headaches, not overdosage to create hallucinations. But just because there are no studies does not mean there are no effects. Anytime one throws the body out of balance with drugs (good or bad) there are effects.\n\nIt was not just hippies that dropped acid, pretty much everybody (people like you and me) from all walks of life experimented with drugs in the 60's 70's and 80's. Hallucinagens were as common then as Ecstasy and Meth are today.\nAs for LSD, the effects are not exactly physical per sey, they occur in the brain and are now described as the phenomenon known as post-hallucinogen perceptual disorder (PHPD). Pretty much flashbacks with a technical name. http://www.erowid.org/psychoactives/health/hppd/hppd_article1.shtml\nI myself can vouch they are real as I know more than one previously bright person who was lost to hallucinagenic overdoses.\n\nLike all things, too much is bad for one...
985648
are you doing a study on the affects on your urine after eating certain substances ? i dont think that was ever done before, but i am not in the habit of sniffing urine on a regular basis.i think you need a hobby.
1029897
http://search.yahoo.com/search?p=%22long+term+effects+of+marijuana%22&ei=UTF-8&fr=ks-ques&x=wrt
62200
ecstasy, is a synthetic entactogen of the phenethylamine family whose primary effect is to stimulate the secretion of and inhibit the re-uptake of large amounts of serotonin as well as dopamine and noradrenaline in the brain, causing a general sense of openness, empathy, energy, euphoria, and well-being. Tactile sensations are enhanced for some users, making general physical contact with others more pleasurable; but, contrary to popular mythology it generally does not have aphrodisiac effects.\n\nshort-term effects\n\nApart from the dangers from impurities, the primary acute risks of taking MDMA are allergic reaction, which is extremely rare, and dehydration. Like amphetamine, MDMA can mask the body's normal thirst and exhaustion responses, particularly if a user is dancing or is otherwise physically active for long periods of time without hydration. MDMA can temporarily reduce the body's ability to regulate its core temperature, and in high-temperature surroundings (e.g. clubs) combined with physical exertion this may lead to hyperpyrexia if precautions are not taken to remain cool. While dehydration is undesirable, there also have been a very small number of users overly concerned about hydration drinking excessive water and suffering from water intoxication and associated hyponatremia (dilution of the blood that can cause swelling of the brain). This is what caused the death of British teen Leah Betts, which may be the world's most widely publicised MDMA-related fatality.\n\nSome users also report decreased libido or impotence; however, studies have had conflicting results\n\nSystemic effects\n\nOther effects include:\n\n    * Pupil dilation with attendant photosensitivity and color perception\n    * Jaw clenching or bruxism ("gurning" or "grinding")\n    * Shutter vision (nystagmus)\n    * General restlessness\n    * Loss of appetite/taste sensation changes\n    * Lack of focus / concentration\n    * Tingling\n    * Sweaty palms\n    * Increased heart rate\n\nVisit the link below to see the slide which displays how ecstacy affects your brain
132405
This is a very useful and infomative site on the general effects of catagorized drugs and individual drugs and the effect on the human body/health.\n\nhttp://teacher.scholastic.com/scholasticnews/indepth/headsup/facts/index.asp?article=medications\n\nHope this helps. I believe it has all the info you are looking for.
208122
Background\n\nThis is a chronic and relapsing condition occurring within apocrine sweat glands, most commonly in women. Deep seated inflammatory nodules form in the axilla, groin, pubic region, and perineum. It is an androgen-dependent disorder and results from keratinous material occluding the sweat pores. There is an association with lithium therapy and Crohn's disease.\n\nEtiology\n\nHS often occurs in multiple members within families, suggesting a genetic predisposition (5). Various etiological agents have been associated. The increased incidence of HS in obese women with acne led to the theory of HS being associated with an hyperandrogenic endocrine disorder (6,7). One study did not find evidence for biochemical hyperandrogenism in women affected with HS (8). Thus, the role of androgens in HS is still not clear. Smoking (9), lithium (10) and oral contraceptives (11) may also be associated with HS, possibly as triggering factors. Nonetheless, the etiology of the disease is unknown. \n\nFrequency\n\nIn the US: In the United States, the prevalence of HS appears to be 1-2% in the general population. \n\nInternationally: The prevalence of HS appears to be 1% of the general population; it was 4% in a group of young adults who were treated at a clinic for sexually transmitted diseases. HS is probably more common than once thought, but the diagnosis is frequently ignored or missed. HS has a worldwide distribution, though hot, humid environments tend to support its development. \n\nMortality/Morbidity\n\nThe death rate is similar to that in the general adult population. \n\nAlthough rare complications of the disease are described, little is known about the typical effects of HS. HS is a chronic disabling disorder that relentlessly progresses and leads to keloids, contractures, and immobility; patients can become outcasts or at least have difficulty in making social contact. \n\nPathogenesis \n\nHistorically, apocrinitis was believed to be the defining histologic feature and pathogenetic mechanism of HS (12,13). Characteristic lobular abscesses in the apocrine gland, demonstrated histologically, seemed to further implicate apocrine gland involvement in the manifestation of disease (12-14). Recently, histological evidence points to HS being a follicular disease (1,15-19). Histological examination in the majority of specimens reveals follicular involvement, including poral occlusion and folliculitis (1). Apocrinitis as the dominant histological feature is found in only a small number of specimens (1,17). Furthermore, a paucity of apocrine glands was demonstrated in the genitofemoral region, one area commonly affected by HS (1). This finding supports the theory that apocrine gland inflammation is not the etiological and pathogenetic mechanism of HS, but rather a secondary manifestation of follicular involvement (1). \n\nClinical Features \n\nHS may arise singularly or multifocally in the genitofemoral, perianal and axillary areas (1-4). The lesions are painful and have a foul odor attributed to bacterial colonization. Erythematous dermal abscesses form that measure up to 2 cm in diameter. Untreated abscesses will gradually increase in size and may drain to the surface. The course of HS is chronic and remitting, with new abscesses arising in previously unaffected areas or in regions of past involvement. Scarring, fibrosis and sinus tract formations are manifestations of late disease. Strictures can occur secondary to sinus tracts; fistulas may also complicate HS. Squamous cell carcinoma is a rare sequella of longstanding HS (20-24). These cancers may be locally aggressive with distant metastases and a high mortality rate (25). \n\nRace/Sex/Age\n\nRace: Although many authors report no racial predilection, an increased incidence is observed in blacks, possibly because blacks have a greater density of apocrine glands compared with whites. \n\nSex: Although HS is widely considered to occur more frequently in females than in males, with
781212
ingestion, inhalation and skin absorption.yes true.skin absorption is observed for chemicals like organophsphorous n they are the most dangerous chemicals.\n\nbtw they are other routs of getting chemicals in body like through injections thats then devided into intramuscular, intra venous, subcutanous, intra dermal etc. still there are some more .
27547
Personal Side Effects\nof Statins\nImpotence, Loss of Libido \nand Erectile Dysfunction \n\n\nThe pharmaceutical industry would lead us to believe that rapidly bottoming out our natural cholesterol levels through the use of their highly touted statin drugs is a relatively innocuous process of definite benefit to society. \n\nBut as we learn more each day of this ubiquitous and unique cholesterol substance, we must question the veracity of their medical advisors. \n\nCholesterol is perhaps the most important substance in our lives for we could not live without an abundant supply of it in our bodies. \n\nResearchers everywhere are learning how extraordinarily complex and often surprising are the pathways that produce and metabolize cholesterol. \n\nCholesterol is the precursor for a whole class of hormones known as the steroid hormones that are absolutely critical for life, as we know it. These hormones determine our sexuality, control the reproductive process, and regulate blood sugar levels and mineral metabolism. \n\nThis same substance that society has been taught to fear happens to be our sole source for our hormones, androgen, estrogen and progesterone.
956482
How about the incidence of eating disorders among young women?  Or the reasons to support use of medicinal cannabis?  Or is world overpopulation real or is it an urban myth?\n\nThere are more suggestions at this site.  Good luck with your speech.
192933
I don't. However it is usually digested into small pieces of paper which is being put on mouth to get into your body.\n\nLSD is an hallucinative, and a very dangerous stuff which can be lethal at the very small amount of overdosing.\n\nIt is reported to damage de cromosones.
266649
yes there is...many studies, one of which is  http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=pubmed&dopt=Abstract&list_uids=3895380&query_hl=3&itool=pubmed_docsum
669796
cant help on the effects part..but here is a link about marijuana and cancer (may pad your report a lil ;-)\n\nhttp://www.sierratimes.com/03/11/07/article_kubby.htm
859649
red eyes\nshortness of breath\nincreased risk of cancer\nshort term memory loss\nlowered sperm count\nerectile dysfunction\nbirth defects\nincreased chances of miscarriage in women\ndifficulty becoming pregnant\ndepression\nincreased chance of mental problems eg paranoid schyzophrenia\npotential jail time\nmonsters in your closet.\n\nKeep in mind the chances of all these things increase with prolonged usage. I have not tried it.  I had to do an assignment on it is high school.  I don't really have a problem with people doing it.  The effects of weed are not that different from the effects of alcohol, except it makes you slower.  Some people are capable of using it in small amounts and others are not capable of controlling the addiction.  It is all dependant on the individual.\n\nI forgot to add ( thankyou Sangre Y Leche) that these are potential side effects and do not necessarily happen to everyone who uses.  The risks are increased with very heavy usage but many people with moderate levels of usage do not display any of the above.
1088522
The University of California Berkeley did a study on the subject.  I think it was in 2004 or 2005.
121658
Bible has a lot of citations about "possessions by demons", and epileptics. I read about the greek doctors in Jesus time, used "beladona", and opium to relieve it. and if you read the "Thousand and one nights", the Arab stories told by princess Scheherezade, that mental illness were quite common and the affected were arrested and locked as animals.
138716
try this report from 1999 to see actual documented studies. http://www.rimbaud.freeserve.co.uk/dhap99f.html
515097
http://www.lef.org/protocols/prtcl-002a.shtml#dhea\n\nI believe in unrefined seas salt. I also found this article verifies what you are saying:\nhttp://www.wholisticresearch.com/info/artshow.php3?artid=78