global glomerulosclerosis?
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Type in glomerulosclerosis on a website like WebMD or eMedicine.  EMedicine is a bit more technical, so that would be a good place to look if you really want to learn about the nuts and bolts of it.  \n\nI can give you a general primer to start your search.  Glomerulosclerosis is the name of a kidney disease.  The filtering structures in the kidney become scarred, and the kidney has less ability to filter the blood.  "Global" means that the scarring affects the entire filter structure, instead of a part of it.  There are thousands and thousands of these structures in the kidney, so the severity of the disease depends on how many are affected.  It's not always clear what causes the disease, but high blood pressure has something to do with the progression of it.  Treatment will involve steroids.  If the kidneys are very badly damaged, transplant might be necessary.  \n\nRemember, these are just the possibilities.  I don't want to scare you with information that might not be relevant to your mother's case.
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GGT has to do with protein synthesis. In terms of steroid use, they increase synthesis. Just a side effect.
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how about dropping all salt intake??  avoid all salty foods.\nif symptoms persist, asked your doc about other higher end anti-biotics.\n\nTIP: AS MUCH AS POSIBLE, AVOID TETRACYCLINE DERIVED ANTIBIOTICS.  THEY CAN CAUSE RENAL DAMAGE. (NEPHROTOXIC)\nI SUGGEST YOU GO FOR A SECOND OPINION. \n\nJOMAR S.T. NDGM '96
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see if these help any...\n\nhttp://www.mypacs.net/cases/AGYRIAPACHYGYRIA-COMPLEX-273885.html\n\nhttp://pathweb.uchc.edu/eAtlas/CNS/669.htm\n\nhttp://rad.usuhs.mil/medpix/radpix.html?mode=single&comebackto=mode%3Dgeo_browse&recnum=767
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Yes.. I have lupus and severe kidney diesease. Had chemotherapy for 3 years, and now on a drug called\n"CELL CEPT".. The both have worked wonders for me.. My kidney function was nearly at 30% three and a half years ago, and now it practically normal again..  Consult a specialist before you try any herbal supplements...... Find a good kidney doc to go to..
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Renal Week 2006 \n\nNovember 14-19 \nSan Diego Convention Center \nSan Diego, California \nhttp://www.asn-online.org/
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Unless one is diabetic, glucose is not lost during nephronic filtering, thus the glucose levels would be equivalent in arteriole and venous blood.  Urea, on the other hand, is filtered out in the kidney (thus excreting waste nitrogen), therefore urea levels in the renal artery would be much greater than that in the renal vein- This is a classic problem of which substances are preferentially filtered in the kidney.  In a person with uncontrolled diabetes there is hyperglycemia, in which the blood glucose levels are much greater then normal-in this case, glucose is filtered out in the kidneys- prior to modern times, physicians used to determine diabetes by tasting the urine of patients to determine if there was sugar in it...
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i have an 8 year old son whom has hypertension as a result of iga nephropathy a very rare condition. he also has a family history of  it.
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Glomerular capillary filtration is sensitive to ionic charge of a compound being passed through it. this is why complex proteins become trapped in the basement membranes and appear as electron dense deposits in tissue sections examined by electronmicroscopy. ionic charge of lipid is repelled
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Mostly an upset stomach or diarrhea.  Long-term overuse has been linked to kidney problems, but nothing definitive.
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juxtaposition juxtaglomerular juxtaposit
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well odd u should ask this my dad has this and thank goodness he went to the dr in time it can cause kidney damage if not treated rapidly...be really careful on your diet not to eat anything with pot,,,rite now till u have been seen by a doc now not to scare u but my dad lost some kidney function he has 37%in one kidney and the other is normal so hes doin just fine goodluck golly email me id like to know how u r doin ladysureone@yahoo.com goodluck
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Chronic renal failure (CRF N18.?) develops slowly and gives few symptoms initially. It can be the complication of a large number of kidney diseases, such as IgA nephritis, glomerulonephritis, chronic pyelonephritis and urinary retention. End-stage renal failure (ESRF) is the ultimate consequence, in which case dialysis is generally required while a donor for renal transplant is found. \nAcute renal failure (ARF N17.?) is, as the name implies, a rapidly progressive loss of renal function, generally characterised by oliguria (decreased urine production, quantified as less than 400 to 500 mL/day in adults, less than 0.5 mL/kg/h in children or less than 1 mL/kg/h in infants), body water and body fluids disturbances and electrolyte derangement. An underlying cause must be identified to arrest the progress, and dialysis may be necessary to bridge the time gap required for treating these underlying causes. \n\nif u still feel lower back pains and difficulty urinating, better go to your doctor for medications
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Most of the sugar and protein constituents that pass through the glomerulus are absorbed back into the circulatory system.  Check out the following diagram on illustration of how the nephron works:\n\nhttp://www1.shore.net/~straub/labsk_nephron.gif
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http://www.webmd.com/content/article/7/1680_53609.htm
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Glycosuria is common in both, but depends on the level. At a plasma glucose up to about 240 mg/dL (give or take a few), the Kidney reclaims all the glucose, so above that, the glucose spills into the urine and drags water with it. In IDDM, glucose easily goes above 240, but in NIDDM, it is possible that the glucose is only slightly elevated, such that glycosuria (and even polyuria) may not happen. This is why NIDDM has more complications at diagnosis - the glucose has been elevated long enough to cause DAMAGE, but not high enough to cause SYMPTOMS. In IDDM, the sugar rises high enough to cause symptoms, and quick enough to diagnose and treat BEFORE complications.\n\nMake sense?
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Try this website. There are loads of information on dialysis, including a message board. Maybe you can meet someone there or find a support group. Good luck!