what is saddle pulmonary embolus?
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A pulmonary embolism is a blood clot that has traveled into the pulmonary (lung) circulation and lodged there, blocking blood flow.  This prevents blood from picking up oxygen at the lungs, so the body tissues can starve from hypoxia.  A saddle pulmonary embolus is probably the worst case scenario.  Whereas most pulmonary emboli are very small (and probably not even clinically significant), saddle emboli lodge where the pulmonary artery divides to the left and right lung.  Blood flow is blocked to both lungs, and death is imminent.\n\nBig emboli like that often form in the large leg veins, especially if someone has been very inactive for an extended period (e.g. a long hospital stay).  Clotted blood isn't the only material that can cause an embolus.  Anything that gets in the bloodstream can cause a blockage -- air, fat, amniotic fluid, bacterial vegetations, and on and on.
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Possibly thrombosis this can be serious .. get it checked out by a doctor regardless of cost!
1134244
Cavernous angioma, also known as cerebral cavernous malformation (CCM), cavernous haemangioma, and cavernoma, is a vascular disorder of the central nervous system that may appear either sporadically or exhibit autosomal dominant inheritance.
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In medicine, pulmonary hypertension (PH) is an increase in blood pressure in the pulmonary artery or lung vasculature. Depending on the cause, it can be a severe disease with a markedly decreased exercise tolerance and right-sided heart failure. It was first identified by Dr Ernst von Romberg in 1891[1]. It can be one of five different types, arterial, venous, hypoxic, thromboembolic, or miscellaneous.\n\nA history usually reveals gradual onset of shortness of breath, fatigue, non-productive cough, angina pectoris, syncope (fainting), peripheral edema, and rarely hemoptysis. Pulmonary arterial hypertension (PAH) typically does not present with orthopnea or paroxysmal nocturnal dyspnea, while pulmonary venous hypertension typically does.\n\nIn order to establish the cause, the physician will generally conduct a thorough medical history. A detailed family history is taken to determine whether the disease might be familial. Physical examination is performed to look for typical signs of pulmonary hyertension including a loud P2 (pulmonic valve closure sound), (para)sternal heave, jugular venous distension, pedal edema, ascites, hepatojugular reflux, etc\nCauses\nThe most common cause of pulmonary hypertension is left heart failure leading to pulmonary venous hypertension. This may be due to systolic or diastolic malfunction of the left ventricle or due to valvular dysfunction such as mitral regurgitation or mitral stenosis. It usually manifests as pulmonary edema.\n\nCommon causes of pulmonary arterial hypertension (PAH) include HIV, scleroderma and other autoimmune disorders, cirrhosis and portal hypertension, sickle cell disease[2], congenital heart disease, thyroid diseases[3], and others. Use of weight loss pills such as Fen-Phen, Aminorex, fenfluramine (Pondimin), and phentermine led to the development of PAH in the past[4]. Other causes include sarcoidosis, histiocytosis X, and fibrosing mediastinitis. Pulmonary embolism also leads to pulmonary hypertension, acutely as well as chronically. When none of these causes can be found, the disease is termed idiopathic pulmonary arterial hypertension (IPAH).\n\nLung diseases that lower oxygen in the blood (hypoxia) are well known causes of pulmonary hypertension, including COPD, interstitial lung disease, Pickwickian syndrome or obesity-hypoventilation syndrome, and possibly sleep apnea. Human herpesvirus 8, also associated with Kaposi's sarcoma, has been demonstrated in patients with PAH, suggesting that this virus may play a role in its development[5].\n\nWhen a family history exists, the disease is termed familial pulmonary arterial hypertension (FPAH). IPAH and FPAH are now considered to be genetic disorders linked to mutations in the BMPR2 gene, which encodes a receptor for bone morphogenic proteins[6], as well as the 5-HT(2B) gene, which codes for a serotonin receptor\nDiagnosis\nNormal pulmonary arterial pressure in a person living at sea level has a mean value of 12–16 mm Hg (1600–2100 Pa). Definite pulmonary hypertension is present when mean pressures at rest exceed 25 mm Hg (3300 Pa). If mean pulmonary artery pressure rises above 30 mm Hg (4000 Pa) with exercise, that is also considered pulmonary hypertension.\n\nDiagnosis of PAH requires the presence of pulmonary hypertension with two other conditions. Pulmonary artery occlusion pressure (PAOP or PCWP) must be less than 15 mm Hg (2000 Pa) and pulmonary vascular resistance (PVR) must be greater than 3 Wood units (240 dyn•s•cm-5 or 2.4 mN•s•cm-5).\n\nAlthough pulmonary arterial pressure can be estimated on the basis of echocardiography, pressure sampling with a Swan-Ganz catheter provides the most definite measurement. PAOP and PVR can not be measured directly with echocardiography. Therefore diagnosis of PAH requires a cardiac catheterization. Swan-Ganz catheter can also measure the cardiac output, which is far more important in measuring disease severity than the pulmonary arterial pressure.\n\nOther diagn
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saddle blocks are used on the area that would hit the saddle on a horse.
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Usually when the reperfussion of the clotted area happens and this can be within the first hour....and sometimes minutes after Retrovase or TPA is started, (clot busters), there will be an arrythmia of several PVCs in a row.  It always makes me catch my breath a bit when I see this run of PVCs.  So far, everytime I've seen this, the irregularity corrects it's self...WHEW!!  Usually within the next 24 hours the person is sent to cath lab for a cardiac cath and depending on what is found and the history of past medical intervention(s)(past by-pass surgery for instance) for cardiac problems, a decision is made to do a by-pass or put in a stent.
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The name for those "bandages" is polo wraps. They can be a long wrap that has to be put on by hand or they can be what is called splint boots. They are there for a few reasons. The first being that they provide support for the area of the leg they are put on. The second is that they provide protection from shoes, kicks, etc, from themselves or another horse. Some horses do what is called over reaching. This happens especially at the trot. The horse will clip his front leg with his back leg and can hurt himself as well as his rider should he fall from it. Third, it can be used to protect minor cuts from being infected by dirt or track sand.   No the horse is not hurt or he would not be racing.
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Since DVT occurs when a thrombus (blood clot) forms in one of the large veins, usually in the lower limbs, leading to either partially or completely blocked circulation. The condition may result in health complications, such as a pulmonary embolism (PE) and even death if not diagnosed and treated effectively.\n\nThis is a question that you really need to ask your Doctor before even considering flying.
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The alveoli is crucial in supplying fresh oxgen which would be transported to the heart via the pulmonary vein.\n\nIs this okay?
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It depends upon where the clot is located in the body - brain, heart, lungs, legs or arms, etc. and how big the clot or occluded vessel is...\n\nClots can lead to pain and tissue death if left untreated.  In the heart or brain they can lead to more rapid deterioration and death.\n\nClots in the leg usually can be caught and treated before they cause problems (move to the lungs, heart, or brain).\n\nBlood clots in the heard, lungs, and brain are the most damaging and life threatening.
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cardiac, smooth, and skeletal.
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It might be a problem with an iron or steel stent.   Ask the doc.
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Pulmonary Edema is an accumulation of serous fluid in the lungs.  After a clot is removed, it is not at all uncommon for serous fluid to accumulate.\n\nSerous fluid, by the way, is caused by the seperation of whole blood when it stagnates after a clot.\n\nWhen the clot is removed, the flow is resumed, and since the lungs have the thinnest membranes, the serum accumulates there.\n\nThis condition is not unusual, and it's easy to correct with surgery.  The risk is minimal and it is not an indication of negligence or malpractice on the doctor's part.  It's just a side effect of the clot.
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The previous answerers did a good job explaining the terminology so I'm going to skip to the significance, which in this case isn't much.  \n\nAccentuated hilar and basal bronchovascular markings is quite a common finding in chest x-rays that could mean anything from normal to very serious disease (if associated with other findings).  It is used as either to confirm a diagnosis by other methods, or as a starting point to investigate further.  \n\nThis finding alone could simply mean that the subject of the x-ray is a smoker, has an acute (e.g. pneumonia) or chronic (e.g. asthma) pulmonary disease, has some degree of valvular heart disease, etc.  Do not read into this more than it merits, but do consult with the doctor that ordered the x-ray to understand what he/she concluded from your work up.
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If you go to a riding stable for a one-hour or afternoon trail ride, you will most likely be riding a horse wearing a Western saddle, who is accustomed to different riders.\n\nMost of the places I visit to do this are fun, because I look for well run, clean barns with well cared for horses and friendly staff.\n\nStay away from any place where the horses look sick and the people are surly.\n\nIf you decide to take riding lessons, some places teach using Western saddles, others use English saddles.  English saddles are smaller, lighter, do not have a saddle horn to hold on to, and you learn to use your legs and your balance to move in rhythm with the horse.\n\nI have taken lessons and leased horses long term.  It's worth the money to go to a well-run place and learn properly.  Then if you want to go for a trail ride, you'll be able to ride at a trot or canter, instead of just a walk.\n\nI always wear a riding helmet.  Even the most experienced riders fall - remember Christopher Reeve?  He was a million times better than I'll ever be.  \n\nAs long as you use common sense and choose your stable carefully, horseback riding is the best fun in the world.
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Medication or surgery. It is very delicate due to the possible release of clots that could cause a stroke or Trans Ischemic Attack (Little stroke)
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http://www.emedicine.com/ped/topic2120.htm