What does Zone of Inhibition mean when dealing with bacteria?
84488
This deals with antibiotic susceptibility testing on (usually Mueller-Hinton) agar plates.  \nFirst you coat the surface of the plate with a bacterial suspension, then you place a disc with whatever antibiotic you're using on the surface of the plate.  After you incubate the plate for about 18 to 24 hours, you will see a zone of inhibition around the disc if the bacteria are killed, or inhibited, by the antibiotic.  This is an area of no bacterial growth.  The sizes of the zones vary depending on the concentration of the antibiotic in the disc, the depth of the agar, the susceptibility of the bacteria, and the concentration of the origional bacterial suspension, etc. . .\n\nHope this helps!
580906
Its a buffer and a metal chelating agent. \nIt prevents metallo proteases and nucleases from funcitoning fully as well as stabilizing the pH of the buffer. \n\nRegards
198655
They block the synthesis of bacteria cell wall\nBeta lactam (contain a ring- beta lactam ring) example are penicillin, cephalosporin, carbapenems\n\n\nSome bacteria now secrete beta lactamase- which stops the beta lactam from working.\n\nHowever, in such infectional bacteria, there are beta lactamase inhibitor that stop the beta lactamase from working. Example: Sulbactam.
1105344
it's important to maintain a certain concentration level of the antibiotic in order to kill the bacteria if you stop the antibiotic it just won't be able to work the next time you need it cause the bacteria is resistant to it. and a vaccine is usually inactivated virus and when they get into your body your body developes antibodies and when you get an active virus the antibodies will fight it
402756
The tendency for hospitals with high prevalences of Extended-spectrum beta-lactamases is to place those patients in similar isolation status as those with vancomycin-resistant enterococci (VRE) or methicillin-resistant Staph aureus (MRSA).  That implies wearing of gloves, gowns, etc., and strict handwashing rules.  \n\nOtherwise, the tendency is to follow the susceptibility results from the cultures and to use narrow-spectrum antibiotics whenever possible.
12095
Sounds like a mild skid infection, that has become locked inside your poars. If the denorex worked, I suggest staying in it awhile longer, say 2 weeks. It killed 98% of the infection, but the remaining 2% grew back. Or you can go see the Doc & get an antibiotic which always works best, but cost more. \nThe 3 key ingredients that promote bacteria growth are "Warmth, moisture, & darkness."
344067
Some Bacteria divide every 20 minutes
559140
depending on the antiseptic you use (if its prescription or over the counter) antiseptic will kill common bacteria and help prevent infections...antibiotic creams depending on what kind you get kill a variety of bacteria but if overused, bacteria can build up resistance and become immune against antibiotics...short term use is safe though...be well!
878427
totally different types of anti-biotics.  you are risking making your infection resistent to the anti-biotics.  i'd call your doc to see what they think you need to do
588318
It breaks down the cell wall of the bacteria.
954483
Antibiotics are making things worse because they have been overused and used to combat viruses, which doesn't work.  The oversued antibiotics kill off the weaker bacteria, leaving room and food for the stronger, more dangerous bacteria that are more likely to cause grave illnesses or kill people.\n\nThe most important means to control infection is, and always has been, frequent hand washing.
658448
Making sure the full course of antibiotics is taken to make sure  all bacteria are killed and none are left to build up resistance. Using a combination of antibiotics is also needed./
270665
C\n\n\nOzone layer is responsible to inhibit UV radiation from Sun to reach earth. Once Ozone layer vanishes, UV radiations are free to reach earth which may cause skin cancer and other diseases.
964493
Microban technology is built-in to products during the manufacturing process and becomes an intrinsic part of the product inside and at the surface. The antimicrobial then works as a cell well penetrant, disrupting key cell functions so that the microorganism is unable to function, grow or reproduce.
490824
hi my name is Geeth Prajwal from India i hope this will help you\n\n\n\nIn order to be useful in treating human infections, antibiotics must selectively target bacteria for eradication and not the cells of its human host. Indeed, modern antibiotics act either on processes that are unique to bacteria--such as the synthesis of cell walls or folic acid--or on bacterium-specific targets within processes that are common to both bacterium and human cells, including protein or DNA replication. Following are some examples. \n\nMost bacteria produce a cell wall that is composed partly of a macromolecule called peptidoglycan, itself made up of amino sugars and short peptides. Human cells do not make or need peptidoglycan. Penicillin, one of the first antibiotics to be used widely, prevents the final cross-linking step, or transpeptidation, in assembly of this macromolecule. The result is a very fragile cell wall that bursts, killing the bacterium. No harm comes to the human host because penicillin does not inhibit any biochemical process that goes on within us. \n\nBacteria can also be selectively eradicated by targeting their metabolic pathways. Sulfonamides, such as sulfamethoxazole, are similar in structure to para-aminobenzoic acid, a compound critical for synthesis of folic acid. All cells require folic acid and it can diffuse easily into human cells. But the vitamin cannot enter bacterial cells and thus bacteria must make their own. The sulfa drugs such as sulfonamides inhibit a critical enzyme--dihydropteroate synthase--in this process. Once the process is stopped, the bacteria can no longer grow. \n\nAnother kind of antibiotic--tetracycline--also inhibits bacterial growth by stopping protein synthesis. Both bacteria and humans carry out protein synthesis on structures called ribosomes. Tetracycline can cross the membranes of bacteria and accumulate in high concentrations in the cytoplasm. Tetracycline then binds to a single site on the ribosome--the 30S (smaller) ribosomal subunit--and blocks a key RNA interaction, which shuts off the lengthening protein chain. In human cells, however, tetracycline does not accumulate in sufficient concentrations to stop protein synthesis. \n\nSimilarly, DNA replication must occur in both bacteria and human cells. The process is sufficiently different in each that antibiotics such as ciprofloxacin--a fluoroquinolone notable for its activity against the anthrax bacillus--can specifically target an enzyme called DNA gyrase in bacteria. This enzyme relaxes tightly wound chromosomal DNA, thereby allowing DNA replication to proceed. But this antibiotic does not affect the DNA gyrases of humans and thus, again, bacteria die while the host remains unharmed. \n\nMany other compounds can kill both bacterial and human cells. It is the selective action of antibiotics against bacteria that make them useful in the treatment of infections while at the same time allowing the host to live another day.
736874
To treat it, not really. Antibiotics are needed.\n\nThere MAY be something to give temporary relief of symptoms, but they wouldn't affect the bacterial.
403949
It is a pretty arbitrary, untestable number, as others said.  It is not clear if they claim it kills that % of actual organisms, or that % of types.\n\nAnyhow, some types are resistant to chemical disinfectants.  These include spores, which have a very resistant coating, able to keep the bug inside alive even in dry conditions etc.  Others are the "acid fast" bacteria, such as Mycobacterium tuberculosis, which causes ... you guesses it... TB!\n\nMicro-organisms compete with each other in various ways, including a type of "chemical warfare", so many are resistant to various chemicals.  A lot of types live in unhospitable and variable environments, eg soil and rocks, too.  They NEED to be tough!\n\nI am a microbiologist.