If someone becomes very ill with pneumococcus pneumoniae, but recovers, would this confer a lifetime immunity?
90277
I believe that there are dozens of serotypes of pneumococcus.  Thus, getting infected with one might only confer immunity to one specific subtype.  The pneumonia vaccine, on the other hand, provides coverage for 20 or so of the most common serotypes.  If you're in a high-risk group (your immune system is compromised or you are elderly), a yearly vaccine would be a good idea.
867868
They catch germs before the germs get into your lungs.  It's easier to get over a sore throat than pneumonia.
363255
It's possible, especially if he sneezed or coughed around you. Just because you recently had an illness doesn't mean that you can't get it again, even soon afterward. This is partly because there are many strains of viruses out there. Your body might have fought something very similar to what your friend got, but that doesn't mean you'll be immune.
631945
Once you have TB you will ALWAYS have it. It is not curable. It can, however, be treated. If it becomes dormant you will not be contagious. Once it is treated it will become encapsulated in what is called a "tubercule" or a "granuloma" where it will remain for the rest of your life, and cause you no symptoms. If your immune system becomes compromised(from cancer, Aids, etc.), then it can break out and become active again. If you have had it once you will always have a positive skin test, and the granuloma will be visible on a chest x-ray. It is, however, very possible to treat it and be symptom free afterward.
506791
it should be fine to mix spiriva with the nebulizer.\n\nYes pneumonia is contagious, but it is LESS contagious than most people think. The cold virus that started the cough in the first place is contagious. But the pneumonia part of the illness down in the lungs is far less contagious. People who are exposed to someone with pneumonia may catch the normal cough and cold symptoms of the virus, but usually don't progress to pneumonia. The formation of the pneumonia is not determined by the particular cold virus or bacteria that the person has, but rather by each person's susceptibility in their own lungs.
508171
Absolutely nil.\n\nIt is possible that birds will develop an immunity over several generations, but that is hardly near future.
782720
I get bronchitis on a regular basis and she needs to drink plenty of fluids, get plenty of rest and take all of the medicine that the Doctor prescribes.  If she's not careful, the bronchitis could progress to pneumonia . . . that's happened to me twice now in the past six years.
358250
Depends what the vacccination is. Some of them are said to have illness connected to them, but a lot of them have not been proved yet.
1054868
The concept of antibiotic immunity is a misnomer that can lead people to believe that they may become "immune" or "resistant" to an antibiotic if they take it too frequently or inappropriately. This notion stems from a misunderstanding in the general public of the concept of antibiotic resistance.\nIt is not people who become immune or resistant to an antibiotic. Rather it is the bacteria responsible for infections that become resistant. The purpose of an antibiotic is to kill--or at least inhibit the growth of--bacteria to quell an infection. Bacteria have been around for thousands of years and have had to survive in hostile environments. As a result, they are able to adapt and have a number of mechanisms for survival, one of which is the development of resistance to antibiotics.\n\nMisuse or overuse of antibiotics selects for resistant bacteria. In a bacterial infection, billions of bacteria are present. Taking an antibiotic will kill or inhibit those bacteria that are susceptible to its effects and leave behind a subpopulation of bacteria that are resistant to the antibiotic. These remaining bacteria can survive and continue to grow. If they then spread from person to person, they may become the norm, and all future infections caused by these resistant bacteria will withstand the original antibiotic, rendering it useless for treatment. For many infections, patients often begin to feel better before all the bacteria have been eliminated. But it is important to complete a full course of antibiotics, even if one’s symptoms have improved, to ensure that as many bacteria as possible are destroyed.\n\nBacteria have a variety of mechanisms by which they become resistant to antibiotics, depending on the particular antibiotic (or class of antibiotic) and the type of bacteria involved. Because antibiotics work differently to attack an essential function of bacteria, it is necessary to consider both the phenotypic mechanism of resistance and the underlying genotypic mechanism of resistance.\n\nFrom a physical or biochemical perspective, there are a number of factors that contribute to antiobiotic resistance. First, the cell wall of a bacterium can be altered so that an antibiotic cannot penetrate it. Second, some bacteria produce enzymes that can destroy antibiotics before they can work. Third, certain bacteria have developed mechanisms known as efflux pumps, which are able to "eject" antibiotics from the bacterial cell before they have a chance to exert any effect. Finally, the antibiotic target site within the bacteria can be altered. In this situation, when an antibiotic enters the bacterial cell it must bind to its intended target in order to have an effect on the bacteria. If the target within the bacteria has been changed to make this binding impossible, the bacteria will be resistant to the medicine’s effects.\n\nUnderlying the phenotypic mechanisms of antibiotic resistance are the genetics of antibiotic resistance. Bacteria, like other living organisms, possess DNA that codes for the proteins and enzymes it requires for survival. Changes to the DNA can result in alterations in the final proteins or enzymes, which in turn can lead to antibiotic resistance. A common genetic mechanism for the development of antibiotic resistance is the acquisition of tiny fragments of DNA known as plasmids or transposons. Transposons may code for a specific antibiotic-inactivating enzyme, which, when produced by the bacteria, destroys an incoming antibiotic before it has an opportunity to work. These tiny fragments may be passed directly from one bacterium to another by a process known as conjugation. Alternatively, small viruses may attack certain bacteria and inject DNA fragments by a process known as transduction. \nAnother genetic mechanism of antibiotic resistance is the acquisition and accumulation of resistance genes from neighboring bacteria through a process known as transformation. A good example of thi
835835
Some organisms are shared between canine and human but this is quite uncommon.  Pneumonia typically occurs only in humans with a weakness in the respiratory system such as asthma or cigarette smoking.  It also tends to occur in the very young (less than 2) and the very 'old' (over 65).  Since you have had pneumonia 4 times - if it was properly diagnosed - there must be a reason.  The likelihood that you or your 2 year old will develop pneumonia with this exposure is exceedingly small - but not impossible.  Thanks for caring for the canine.
67311
Pneumonia (say: new-mo-nya) is an infection of one or both lungs. To know what that means, you have to know something about lungs and what they do. When you breathe in, you pull oxygen into your lungs. That oxygen travels through breathing tubes and eventually gets into your blood through the alveoli (say: al-vee-oh-lie). \n\nAlveoli are tiny air sacs covered in tiny blood vessels called capillaries. How can something so small get oxygen into your blood? You have about 600 million of them! When oxygen-rich air reaches the alveoli, it can be absorbed into the blood. Then your red blood cells can carry oxygen all over your body. The body needs oxygen to keep working properly and to stay alive. \n\nBut if a person has pneumonia, his or her lungs can't do this important job very well. Why? Because this kind of infection creates fluid that blocks the alveoli. This makes it hard for oxygen to get deep into the lungs, where it can be passed through to the blood. The person can still breathe, but it might be hard to breathe, especially if the pneumonia affects both lungs.\n\nPneumonia can happen to people at any age, from tiny babies to really old people. Getting wet doesn't cause pneumonia, but an infection from bacteria or a virus does. A cold or flu that gets worse can turn into pneumonia. That's because the cold or flu will irritate the lungs, creating an environment where it's easier for pneumonia germs to move in and start an infection.
654149
It depends on a few things: how old you were when you had it, how severe your case was, and how healthy you are now.  If you were very young (like, 2 years or younger) it is possible that your body didn't make sufficient antibodies for your adult life.  If you didn't have a very serious case, the same may be true.  If you have a compromised immune system for any reason, you could also be vulnerable.\n\nHowever, if you are healthy, had a pretty solid case previously, and were at least old enough to remember being sick, you're probably okay.  Still, I wouldn't suggest going out of your way to be around your roomates' things.  Wash your hands, get lots of rest, drink lots of fluids - play it safe.
346531
When you ask does pneumonia turn toxic, what do you mean?  If you are refering to a person with pneumonia going into septic shock it is possible depending on the circumstances.\n\nPneumococcal pneumonia is often preceded by a URI. The onset is often sudden, with a single shaking chill; persistent chills suggest another diagnosis. The chill is ordinarily followed by fever, pain with breathing on the affected side (pleurisy), cough, dyspnea, and sputum production. The pain may be referred and, with lower lobe involvement, may suggest intra-abdominal sepsis, such as appendicitis. The temperature rises rapidly to 38° to 40.5° C (100.4° to 105° F), the pulse is usually 100 to 140 beats/min, and respirations accelerate to 20 to 45 breaths/min. Additional common findings are nausea, vomiting, malaise, and myalgias. The cough may be dry initially but usually becomes productive with purulent, blood-streaked or rusty sputum. In many patients, especially those at age extremes, symptoms may be more insidious.\n\nFindings during physical examination vary depending on the character of the process and the stage in which the patient is evaluated. There may be typical signs of lobar consolidation or pleural effusion. In bronchopneumonia, the usual finding is crackles.\nComplications\n\nSerious and potentially lethal complications include progressive pneumonia, sometimes associated with the adult respiratory distress syndrome and/or septic shock. Some patients develop infections at contiguous sites (eg, empyema or purulent pericarditis). Pleural effusions are found in about 25% of patients by chest x-ray, but < 1% have empyema. Bacteremia may result in extrapulmonary infections, including septic arthritis, endocarditis, meningitis, and peritonitis (in patients with ascites). Some patients develop pulmonary superinfections: temporary improvement during treatment is followed by deterioration, with recurrence of fever and worsening pulmonary infiltrates.\nlthough the morbidity and mortality of pneumococcal pneumonia have decreased substantially since the advent of penicillin, pneumococcus accounts for about 85% of all lethal cases of community-acquired pneumonia in which an etiologic agent is known. The overall mortality rate is about 10%, and treatment has minimal effect on mortality during the first 5 days of illness. Factors that herald a relatively poor prognosis include age extremes, especially < 1 yr or > 60 yr; positive blood cultures; involvement of > 1 lobe; a peripheral WBC count < 5000/µL; presence of associated disorders (eg, cirrhosis, heart failure, immunosuppression, agammaglobulinemia, anatomic or functional asplenia, and uremia); involvement of certain serotypes (especially 3 and 8); and development of extrapulmonary complications (eg, meningitis or endocarditis).\n\nMildly ill patients who are treated relatively early usually defervesce during the first 24 to 48 h; however, seriously ill patients, particularly those with the poor prognostic features noted above, often require >= 4 days to become afebrile. Therapy should not be modified if there is gradual clinical improvement and the etiology is confirmed.\n\nWhen patients do not improve, these factors should be considered: wrong etiologic diagnosis, adverse drug reaction, far-advanced disease (most common), superinfection, inadequate host defenses due to associated conditions, noncompliance with the drug regimen by outpatients, antibiotic resistance of the involved strain of S. pneumoniae, and complications, such as empyema requiring drainage or metastatic foci of infection requiring a higher dosage of penicillin (eg, meningitis, endocarditis, or septic arthritis).
457389
By getting sick. Most likely pnuemonia.
100739
No one can accurately predict whether it will or will not reoccur. Allergist are notoriously vague about this issue.\nMy daughter had severe asthma every winter for 5 years. We ended up in the hospital every winter.I removed carpeting, stuffed animals, covered mattresses,put air purifiers and filters in our home, Changed detergent, got allergy shots for 4 years.  So far, she's much better.  But the doctors are hedging their bets.
121978
You would become very sick. (Sicker according to the question)\nYou would become anemic.
838023
I can't see a relationship, a pneumonia affects your lung whereas a stroke affects your brain. You might develop pneumonia if you're lying in bed for too long, which can happen after a stroke.\nEspecially the elderly. If paralized after a stroke and bedridden, the patient needs to be turned on his/her side regularly.