what are the long term effects from trichinosis?
81313
Hi rubymaroon,\nTry http://www.webmd.com/. Good Luck: Blueladybug
398303
Nothing much. Only vomitting, Nausea, fever, headache, weakness will be the minor side-effect. At the end, everything will be normal and you will also lose weight.
261897
First you get didarrhoea and dysentery.  In the long run you get , Gastritis, Peptic ulcers, stomatitis,etc.
1115263
Health effects\nThe major concern is that there are many sensitive land uses where people are in direct contact with soils such as residences, parks, schools and playgrounds. Other contact mechanisms include contamination of drinking water or inhalation of soil contaminants which have vaporized. There is a very large set of health consequences from exposure to soil contamination depending on pollutant type, pathway of attack and vulnerability of the exposed population. Chromium and many of the pesticide and herbicide formulations are carcinogenic to all populations. Lead is especially hazardous to young children, in which group there is a high risk of developmental damage to the brain and nervous system, while to all populations kidney damage is a risk.\n\nChronic exposure to benzene at sufficient concentrations is known to be associated with higher incidence of leukemia. Mercury and cyclodienes are known to induce higher incidences of kidney damage, some irreversible. PCBs and cyclodienes are linked to liver toxicity. Organophosphates and carbamates can induce a chain of responses leading to neuromuscular blockage. Many chlorinated solvents induce liver changes, kidney changes and depression of the central nervous system. There is an entire spectrum of further health effects such as headache, nausea, fatigue, eye irritation and skin rash for the above cited and other chemicals. Clearly at sufficient dosages a large number of soil contaminants cause death.
237919
Exposure : \n  \nThe inorganic compounds do not penetrate the body via skin like some organic compounds, such as certain \ntricarbonyls. \n  \nInhalation of dust or fume is the major route of entry in occupational manganese poisonning. Also inhaled large particles are ingested after mucociliary clearance from the lungs. Gastrointestinal absorption is generally low (5%). Very few poisonings have occured after ingestion. \n  \n  \nAcute intoxication : \n  \n1 - Metal fume fever : \nInhalation of manganse oxide fumes may cause a flu-like syndrome similar to " metal fume fever ", treatment is \nsymptomatic. \n  \n2 - Chemical pneumonia : \nIn the case of severe exposure to fumes or dust of various manganese salts, a severe chemical pneumonia may occur. \n  \n3 - Acute intoxication by ingestion : \nAcute intoxication by ingestion rarely occurs and is caused by accidental or voluntary ingestion of a manganese salt (as the ingestion of tablets of potassium permaganate), this chemical causes massive burns of the digestive tract, oedema of the upper respiratory tract and circulatory collapse. \n  \nChronic intoxication : \n  \nThe primary target organ of manganese toxicity is the central nervous system, particularly the extra-pyramidal system ; the lungs may also be injuried in the case of chronic exposure to manganese. \n  \n1-Central nervous system : \n  \nNeurological symptoms are caused by inhalation of fumes or dusts of manganese. \nThe extra-pyramidal system is the main target organ of manganese. \nExposure to heavy concentrations of dusts or fumes for as little as three months may produce the condition, but usually cases develop after 1-3 years of exposure. \nIn manganese mines where most cases have occurred, the disease has appeared after 10 to 20 years of exposure. \nThe symptoms may simulate progressive bulbar paralysis, postencephalitic Parkinsonism, multiple sclerosis, \namyotrophic lateral sclerosis and progressive lenticular degeneration(Wilson's disease). \n  \nThe best way to diagnose, at an early stage, manganese intoxication is neurological examination. \nA standardized questionnaire of neurological symptoms is helpful. \n  \nHere are some symptoms, among others, to be looked for in chronic manganese intoxication : \n  \n- nervousness \n- irritability \n- memory loss \n- tiredness \n- insomnia \n- muscle weakness \n- muscle pain \n- trembling fingers \n- stiffness of limbs \n- difficulty with fine movements \n- stuttering \n- hoarse voice \n- urinary problems \n- impotence. \n  \nAt physical examination your doctor should look for signs of an extra-pyramidal syndrome at its beginning. \n  \nLaboratory and diagnostic testing in manganese toxicity: \n  \nLaboratory or diagnostic test \n Abnormal findings \n \nUrinary levels \n >10 mg/L (normal reference, range 0.5 to 9.8 mg/L; up to 50 mg/L for occupational exposure) \n \nWhole blood levels \n >19 mg/L (normal reference, range 8.0-18.7 mg/L) \n \nSerum levels \n >1.3 mg/L (normal reference, range, 0.3-1.3 mg/L \n \nElectroencephalographic parameters  Low-amplitude, weakened rhythms \n \nNeuropsychiatric testing \n Decreased memory, decreased reaction time, decreased motor coordination \n \nCT scan, magnetic resonance imaging \n Radiodense accumulations of manganese in affected areas, abnormalities in globus pallidus, caudate nucleus, putamen \n \nPositron emission tomography \n Normal fluorodopa scan \n \nTreatment : \n  \nAt the beginning of the intoxication, it seems that a chelation treatment with calcium EDTA may favor an improvement of the clinical picture. At an advanced stage of the intoxication this treatment is ineffective. In this case, the treatment is identical to Parkinson's disease (levodopa).The usefulness of this treatment is however contoversial.
1110021
serious side effects, \n\nan allergic reaction (difficulty breathing; closing of the throat; swelling of the lips, tongue, or face; or hives); \nan irregular or fast heartbeat; \nchest pains or very high blood pressure (blurred vision, severe headache, flushing); \nunusual behavior or confusion; or \nliver damage (yellowing of the skin or eyes, nausea, abdominal pain or discomfort, unusual bleeding or bruising, severe fatigue).
995431
You are not likely to be exposed to large enough amounts of cyanide in the environment to cause adverse health effects. The severity of the harmful effects following cyanide exposure depends in part on the form of cyanide, such as hydrogen cyanide gas or cyanide salts. Exposure to high levels of cyanide for a short time harms the brain and heart and can even cause coma and death. Workers who inhaled low levels of hydrogen cyanide over a period of years had breathing difficulties, chest pain, vomiting, blood changes, headaches, and enlargement of the thyroid gland.\nthe answer to you question depends an the amount, did the person die from cyanide? tobacco smoke is the most common source of cyanide exposure for the general population. So no, your lungs aren't going to anyone, but other parts might. But if it's hydrogen cyanide or cyanide salts these produce spores, and no one is going to want anything from someone with these kinds of spores
405069
Honestly, the effects are endless. I'm currently working on this topic for my Master's (that what I should be doing right now) and when you really start to delve into the study of it, it's really quite disgusting because it affects everything we rely on to live.  But to sum it up, when the land is polluted, it really leads to pollution, destruction, and loss of everything else. The pollution makes it way into our water sources, kills plants, and  consequently, that is one cause that leads to the loss of animal species.  Polluted land is often treated with chemicals to "clean it up" that ends up in the soil and poisons our food sources.  The issue really is complex, but I encourage you to study it further.  You won't look at this world the same.
627382
not good. the nicotine affects ur liver and respiratory system in the long run
1142305
the main side effect is tachycardia which is rapid heartbeat. Go to http://www.nlm.nih.gov/medlineplus/ency/article/002636.htm#Symptoms for advice if you have such a patient, and as soon as possible. That website is a national N.I.H. (U.S.A.), poison information site.\nYou should answers for the following questions before the contact.\nthe patient's age, weight, and condition \nthe name of the product (ingredients and strengths if known) \nthe time it was swallowed \nthe amount swallowed \nAlso contact the local poison control center.\nGood luck
1016843
Nausea and vomiting are the main ones. Of course you can also have the traditional side effects or oral contrceptives such as blood clots, spotting, etc.
624644
Potential Health Effects -Ingestion:Large oral doses may cause irritation and blistering to the mouth, throat, and abdomen. May also cause abdominal pain, vomiting, and diarrhea. if you didnt swollow a lot i thin kyou'll be fine but to be safe id drink a lot of water to help flush it out
620289
Possible side effects include:\n\nheadache, agitation, depression, fatigue, insomnia, restlessness, dyphonia (Difficulty in speaking), horseness, throat infections, cataracts, nasal stuffiness/sinusitis, brochospasm, cough, wheezing, dry mouth, dyspepsia (Upper abdominal discomfort, often chronic or persistent, colloquially referred to as "indigestion." ), and nausea\n\nNo urinary hesitancy listed
53913
It's not really scabies ( tiny insects which burrow along in skin and lay eggs as they go) that cause the damage,  it is the subsequent scratching, itching, and digging that causes problems.  Repeated scratching can lead to skin infections.  Since skin is the largest organ in the body, infections can be extremely dangerous. Untreated skin infections can lead to blood infections or kidney infections.  If your lucky enough to avoid these problems, scarring of the skin from continual scratching is something to look forward to.  In short-see your local pharmacist for an over the counter medication to treat scabies.  Stay healthy!
335676
I haven't heard of any side effects. You could try Google?
390418
Eating food or drinking water contaminated with high levels of creosotes may cause a burning in the mouth and throat, and stomach pains.\n\nBrief direct contact with large amounts of coal tar creosote may result in a rash or severe irritation of the skin, chemical burns of the surfaces of the eyes, convulsions and mental confusion, kidney or liver problems, unconsciousness, and even death. Longer direct skin contact with low levels of creosote mixtures or their vapors can result in increased light sensitivity, damage to the cornea, and skin damage. Longer exposure to creosote vapors can cause irritation of the respiratory tract.\n\nLong-term exposure to low levels of creosote, especially direct contact with the skin during wood treatment or manufacture of coal tar creosote-treated products has resulted in skin cancer and cancer of the scrotum. Cancer of the scrotum in chimney sweeps has been associated with long-term skin exposure to soot and coal tar creosotes. Animal studies have also shown skin cancer from skin exposure to coal tar products.\n\nThe International Agency for Research on Cancer (IARC) has determined that coal tar is carcinogenic to humans and that creosote is probably carcinogenic to humans. The EPA has determined that coal tar creosote is a probable human carcinogen.\n\nThere is no unique exposure pathway of children to creosote. Children exposed to creosote will probably experience the same health effects seen in adults exposed to creosote. Children who played on soil contaminated with creosote had more skin rashes than children who played in uncontaminated areas. It is unknown whether children differ from adults in their susceptibility to health effects from creosote.\n\nStudies in animals have shown birth defects in the young of mothers exposed to high levels of creosote during pregnancy, but no similar studies have been performed on humans. Some animal studies indicate that creosotes may cross the placenta and reach the fetus. Because chemical components (PAHs, cresol, phenols) of coal tar creosote may be stored in body fat, they may be found in breast milk and could pass to nursing infants.\n\nIn 2003 the European Union banned creosote for amateur and unlicenced professional use, due to concerns over these health effects, noting that recent research had shown that the risk of skin cancer had perhaps been underestimated previously.
152828
Hmm, AMONG  ALL KINDS OF POSSIBLE INFECTIONS, YOU MUST BE CAREFUL ABOUT TETENUS TOXID BACTERIA, YOU MIGHT NEED TO TAKE A VACCINATION SHOT (IF YOUR LAST VACCINATION SHOT WAS MORE THAN 10 YRS)Tetanus is a serious and often fatal disease caused by the neurotoxin tetanospasmin which is produced by the Gram-positive, obligate anaerobic bacterium Clostridium tetani. Tetanus also refers to a state of muscle tension.\n\nBacilli of C. tetani can be found in soil (especially agricultural soil), and the intestines and feces of horses, sheep, cattle, rats, dogs, cats, guinea pigs, and chickens. Spores are found in manure-treated soil, skin surfaces (of both animals and humans), under nail-beds, and in contaminated heroin.\nSymptoms\n\nThe incubation period for tetanus is 3 days to as long as 15 weeks (with the average being about 8 days) [2]. For neonates, the incubation period is 4 to 14 days, with 7 days being the average. Most of the time, the further the wound is from the central nervous system, the longer the incubation period. Incubation period length and likelihood of death are inversely proportional; a deep, contaminated wound that allows the bacteria to flourish and causes a quick, aggressive infection is much more life-threatening than a shallower, less-contaminated wound that causes milder symptoms to appear days or weeks later.\n\nThe first sign of tetanus is a mild jaw muscle spasm called lockjaw (trismus), followed by stiffness of the neck and back, risus sardonicus, difficulty swallowing, and muscle rigidity in the abdomen. The stiffness and spasming of muscles expands throughout the body inferiorly, and can be so powerful that they cause muscle tears and even fractures. Typical signs of tetanus include an increase in body temperature by 2 to 4°C, diaphoresis (excessive sweating), an elevated blood pressure, and an episodic rapid heart rate. Spasms and muscle contraction last for 3 to 4 weeks, and complete recovery may take months. About 30% of tetanus victims die, most of whom are elderly patients. In developing countries, the mortality rate may be as high as sixty percent.\n\nComplications of the disease include spasms of the larynx (vocal cords), accessory muscles (chest muscles used to aid in breathing), and the diaphragm (the primary breathing muscle); fractures of long bones secondary to violent muscle spasms; and hyperactivity of the autonomic nervous system.\nhttp://www.freeinfosociety.com/site.php?postnum=342