alzheimer s disease-  is there a cure based on hormonal replacement?
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alzheimer(idopathic disease) .up till now .there are 2 drugs that help to slow down mental deterioration in early stages, but still no cure for alzheimer is found .
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From Mens Health and Well Being Association\n\nTestosterone is not just a sex hormone.  Every cell in the body has a receptor for testosterone. It is vital in the production of protein and therefore in the formation of muscle. It has a key role in the manufacture of bone. It improves the oxygen uptake of cells, helps control blood sugar, helps regulate cholesterol levels and helps maintain a powerful immune system. It helps concentration, improves mood and may protect against Alzheimer’s disease.\nThe male form of menopause is called andropause and is caused by a slow decline in testosterone production by the testicles and adrenal glands.  In men, 90-95% of testosterone is produced by the Leydig cells of the testicles and the remainder is formed by the adrenals. Unlike the female menopause, which is caused by a sudden decrease in hormone levels, the male andropause occurs much more gradually over a 20 year period and therefore may be less obvious.\n\nPeak testosterone levels in men occur in the late teens. After that there is a gradual decline. Andropause typically begins in men in their early forties or fifties. By the age of 50, 50% of men have low testosterone levels compared to age based guidelines. That figure is a lot higher if levels are compared to optimal levels that occurred during youth. By the age of 60 at least 50% of men will have levels of testosterone low enough to start causing serious health problems. A few men are lucky enough to maintain youthful levels right into old age and the result is usually health and vitality. For the rest of us, symptoms of andropause can include:\n \n\n    * fatigue (often the earliest sign). You just don’t feel like getting up in the morning anymore. It’s called the TAT syndrome (Tired All the Time).\n    * depression, anxiety and irritability. It’s called the USTA syndrome (I ‘usta’ enjoy life).\n    * reduced libido\n    * decreased morning erections\n    * loss of drive, creativity and self-confidence\n    * facial wrinkles\n    * muscle pain and stiffness\n    * increased body fat\n    * decreased muscle mass\n    * increased blood pressure\n    * angina\n\n\nIt is associated with an increase in the incidence of heart attacks, strokes and brain ageing.  There is also a strong association between decreased testosterone levels and type 2 diabetes, elevated cholesterol, osteoporosis and arthritis.\n\nSo how do you know if you would benefit form testosterone replacement? First of all, check to see if you have any of the above symptoms.
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Maffei, et al investigated whether estrogen therapy exerted vasoactive affects on post menopausal, healthy women.  Their results, "obtained in healthy women in recent menopause, indicate that the ratio between vasodilator (NOx and prostacyclin) and vasoconstrictor (Estrogen Therapy) bioavailability shifted towards the previous ones after Oral-ET, while it remained unchanged after Transdermal-ET; moreover, catecholamines levels were reduced by both treatments already from 1 month of therapy. These changes might represent very early beneficial effects evoked by ET on the cardiovascular system."\n\nHwang et al investigated the long term effect of estrogen replacement on plasma nitric oxide levels and the effect of arteriosclerotic plaquing.  "In this study, we found no evidence for an estrogen-induced effect on plasma total NO levels which unlikely accounted for the mechanism underlying the 17beta-estradiol atheroprotective effect on subclinical atherosclerosis progression."\n\nKonukoglu, et al investigated plasma total homocysteine concentrations in obese and non-obese female patients with type 2 diabetes mellitus; its relations with plasma oxidative stress and nitric oxide levels.  They determined "there was no significant correlation between plasma homocysteine and plasma NOx, insulin and blood pressure. They thought that homocysteine might have a permissive role on the endothelium damage through free radical generating systems and the presence of obesity the free radical induced-damage has been elevated in diabetic patients."\n\nR.W. Ke et al was investigating Effect of short-term hormone therapy on oxidative stress and endothelial function in African American and Caucasian postmenopausal women.  In addition, HT's effect on plasma nitric oxide (NO) activity was determined as a measure of vascular endothelial function. They investigated the relationship of these markers and HT across race and the cardiovascular risk factors of smoking, diabetes and hypertension.\nThey determined that "Nitrite, the stable oxidation metabolite of NO, was measured by the Greiss reaction after nitrate reduction to nitrite with cadmium.Plasma levels of free 8-isoprostane decreased significantly after 6 weeks of HT. Although almost all subjects benefited from the reduction in free 8-isoprostane, PMW with at least one cardiovascular risk factor (n = 19) demonstrated higher free 8-isoprostane than did subjects with no risk factors. "\n\nIn sum it appears that estrogen in the form of oral hormone replacement therapy increases the plasma nitrite levels in diabetic, post menopausal females to a greater extent than that of their non-cardiac risk factor counter parts.
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Hormones don't help lose weight. A proper diet and the proper physical exercise does. Visit a personal trainer for the plan to best suit your needs based on your weight and active levels.\n\nAs far as regaining libido during menopause, estrogen re-uptake would be your best option here. I recommend looking into vitamins, most commonly labeled as Premarin or Cenestin. These vitamins will greatly aid during menopause. Please consult with your family physician for more information.\n\nBest of luck.
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Alzheimer’s disease\nDefinition \nAlzheimer's disease (AD), one form of dementia, is a progressive, degenerative brain disease. It affects memory, thinking, and behavior. \n\nMemory impairment is a necessary feature for the diagnosis of this or any type of dementia. Change in one of the following areas must also be present: language, decision-making ability, judgment, attention, and other areas of mental function and personality. \n\nThe rate of progression is different for each person. If AD develops rapidly, it is likely to continue to progress rapidly. If it has been slow to progress, it will likely continue on a slow course. \n\nAlternative Names \nSenile dementia/Alzheimer's type (SDAT) \n\nCauses \nMore than 4 million Americans currently have AD. The older you get, the greater your risk of developing AD, although it is not a part of normal aging. Family history is another common risk factor. \n\nIn addition to age and family history, risk factors for AD may include: \n\nLongstanding high blood pressure \nHistory of head trauma \nHigh levels of homocysteine (a body chemical that contributes to chronic illnesses such as heart disease, depression, and possibly AD) \nFemale gender -- because women usually live longer than men, they are more likely to develop AD\nThere are two types of AD -- early onset and late onset. In early onset AD, symptoms first appear before age 60. Early onset AD is much less common, accounting for only 5-10% of cases. However, it tends to progress rapidly. \n\nThe cause of AD is not entirely known but is thought to include both genetic and environmental factors. A diagnosis of AD is made based on characteristic symptoms and by excluding other causes of dementia-.
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Not that I have ever heard ... cat litter has been associated (rightly or wrongly) with Alzheimers, however. There is small evidence (disputed) that Alzheimers is linked to increased levels of aluminum in the body. Cat litter can contain aluminum oxide.\n\nI personally think this is one of those scare stories put about by people who only half read the watered-down media redactions of medical research.
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First of all are you referring to Perimenopause? The most common treatment for perimenopause involves the use of oral contraceptives or hormone replacement therapy. The low-dose pills that are available today regulate menstrual flow and frequency. They also can eliminate or reduce hot flashes, vaginal dryness, and emotional and physical symptoms of premenstrual syndrome. Dietary changes may also help. Women in perimenopause will benefit from a diet high in calcium, low in fat, and rich in fruits, vegetables, and whole grains. This healthful diet helps prevent osteoporosis, heart disease, and some cancers. It may also help reduce symptoms of perimenopause. Exercise helps control weight, improve sleep, and keep bones strong. Exercise also helps with mood swings. Thirty minutes of exercise on most, if not all, days is recommended for everyone.
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It is normal to prescribe low-dose hormones or low-dose hormone birth control pills to women going through menopause to relieve some of their symptoms, such as hot flashes.  \n\nThere have been a variety of studies questioning the safety of hormonal treatment for women.  There is a link below providing a summary of some of these issues.\n\nIf you are concerned, you should discuss this with your doctor or seek a second opinion.\n\nI have also included a link to information about premature menopause, generally defined as menopause occuring in women under the age of 40.  There is no way to reverse menopause once the process has started, so the birth control pills your doctor prescribed are presumably meant to help with your symptoms.
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I don't think so. Mental illness displays lack of or excess activity in certain areas of the brain, usually caused by changes in hormone levels. If the brain modified its structure over the years, would medications be able to make the illness manageable?
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Hormonal activity is usually a negative feed back mechanism. You would think exercise would feed forward, though, as the rate would keep increasing. Not my area, though, so a functional biologist is needed here.
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There are many, check out the website below.  It depends on if you are looking for someone to treat an Alzheimer's patient, or if you are looking for someone who does basic science research into its causes.  Look for people at Alzheimer's disease centers (second link)
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*Hunting a good source* Alzheimer’s is the most common form of dementia and accounts for at least half of all dementias. It causes about 23,000 deaths a year in the United States, making it the eighth leading cause of death in the elderly population. Alzheimer's disease is named after Alois Alzheimer, the German physician who identified it in 1907. The cause of Alzheimer's remains elusive and resistant many treatments.\n\nPeople with Alzheimer's disease have an atrophy of the cortical tissue within the brain. When examined at autopsy the brain shows abnormal fibers that appear to be tangles of brain tissue filaments (neurofibrillary tangles) and senile plaques (patches of degenerative nerve endings). This damage is believed to cause disruption to transmission of impulses amongst brain cells. It is this damage that is thought to produce the symptoms of the disease.\n\nPeople over the age of 65 are most frequently affected by Alzheimer's and it is termed ‘late onset’. So-called ‘early onset’, a very rare form of Alzheimer's disease can develop in people as young as 40 years and in middle age.\n\nAlzheimer’s begins with mild memory problems, attention lapses and difficulty in recalling words thus making communication more difficult. As the disease progresses simple tasks become difficult or impossible to achieve. Distant memories begin to fade, familiar faces often go unrecognised and changes in personality become noticeable.\nFor more see http://alzheimers.about.com/cs/diagnosisissues/a/what_alzheimers.htm
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Sex Rx for Women\n\nIf you're a woman with a low sex drive, you're probably asking, "Where's my pill?" The short answer is, don't hold your breath—but don't give up hope, either. The most promising treatments:\n\n>Viagra The Viagra-for-women idea isn't new: Irwin Goldstein, M.D., director of the Institute for Sexual Medicine at Boston University School of Medicine, found it enhanced clitoral blood flow in rabbits way back in 1998. More recently, a study in the European Journal of Obstetrics and Gynecology found that women treated with Viagra for four weeks reported marked improvement in arousal, orgasm, and sexual enjoyment (side effects were similar to those reported in men). So why aren't women hoarding their partners' pills?\n\n"Drugs for erectile dysfunction work well for many men because they typically want to have sex but can't," Goldstein explains. "For women there's often a different problem: In many cases what's missing is the desire." A woman's sexuality is more complicated than a man's—big surprise there—which means that boosting desire may require something other than improving circulation. For women, Viagra will likely have a better success rate for lubrication problems than libido problems, since these are less about blood flow and more about hormones and psychology.\n\n>Estrogen Therapy It's well known that estrogen shortages can lower libido and trigger such problems as vaginal dryness. Events such as childbirth, hysterectomy, and menopause can all cause imbalances, but so can drugs like antidepressants and, ironically, birth control pills. If eliminating problematic prescriptions doesn't correct estrogen imbalances, a woman may need estrogen replacement. For women who've been rattled by the recent controversy over estrogen pills and patches, alternatives include locally applied creams like Premarin and Estrace, and vaginally inserted drugs like Estring and Vagifem.\n\n>Androgen Therapy Estrogen imbalance isn't even half the problem, explains Goldstein. "There are 10 sex steroids in all, and only three are estrogens; the other seven are androgens, which most people think of as the male sex hormones. We're finding that three out of four women who see us for lack of desire have serious androgen shortages."\n\nTherapy typically focuses on upping female levels of testosterone. For women, topical formulations are the most common delivery method. Creams have been marketed for men for years, but gels such as Testim and AndroGel get top marks from Goldstein. The big reason: They're more effective and much easier to use. Androgens are also available in pill form—one product, Estratest, combines a type of testosterone with estrogen—but the most exciting news may be the buzz about dehydroepiandrosterone (DHEA), a chemical cousin of estrogen and testosterone. Goldstein's research has found that women who take DHEA show substantial improvements in their self-evaluated sexual performance—though he's quick to warn that women shouldn't take it on their own. "It's sold as a supplement, but DHEA is a drug, and we don't totally understand it yet," explains Goldstein. While side effects of topical and oral testosterone therapy can include weight gain, clitoral enlargement, and unwanted hair growth, DHEA use has been linked to even scarier problems including birth defects and cancer.\n\n>Talking Cure A wide range of psychological factors, including depression, anxiety, a history of physical and emotional abuse, and an array of interpersonal relationship problems, can drain a woman's interest in sex. "While we never want to say that problems with sex are all in someone's head, the psychological factors are undeniable and may even be more profound in women than they are in men," says Goldstein. That's the reason all of his patients consult with
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yeah legal dude:)\n\n\nits called bio-identical hormone replacement,\n\nalso    progenelone   , dhea, increase testorone   because are \nprecursor,\n\nexercise\n\nand fasting also increases all hormones
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No, though doctors are using Aricept nowadays to slow the dementia process. \n\nFun Fact (if you can call Alzheimers fun): Did you know that the only way to truly diagnose Alzheimers is with an autopsy and examination of the brain? When doctors make the diagnosis of Alzheimers they are only making an educated guess.
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Alzheimer's disease (AD), a neurodegenerative disease, is the most common cause of dementia and characterized clinically by progressive cognitive deterioration together with declining activities of daily living and neuropsychiatric symptoms or behavioral changes. The most striking early symptom is memory loss (amnesia), usually manifests as minor forgetfulness that becomes steadily more pronounced with illness progression, with relative preservation of older memories. As the disorder progresses, cognitive (intellectual) impairment extends to the domains of language (aphasia), skilled movements (apraxia), recognition (agnosia) and those functions (such as decision-making and planning) closely related to the frontal and temporal lobes of the brain as they become disconnected from the limbic system, reflecting extension of the underlying pathological process. This consists principally of neuronal (cell) loss (or atrophy), together with an inflammatory response to the deposition of amyloid plaques and neurofibrillary tangles.
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The GNP gains from the cell phone industry are so massive that there was no time for study of longitudinal effects. There is some evidence of non-thermally caused activation of HSP protein (chaperonins), and also minor increases in short term memory for males, but not females. Calcium-ion channel alterations in neurons were originally suspected but have recently been shown not to occur. The message to leave you with is definitively not to allow children to use them as the growing brain is much, much, much more vulnerable than the adult brain. But, a direct link to cancer has not been shown.\n\nConcerned about gridlock? Google "Hallitubes" and write us what you think: hallitubevolunteer1@yahoo.com